Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster Display session

360P - Lymphocyte activation gene-3 (LAG-3) and cluster of differentiation-155 (CD-155) expression by immunohistochemistry do not predict response and survival in non-small lung cancer (NSCLC) patients treated with second-line nivolumab treatment

Date

28 Mar 2025

Session

Poster Display session

Presenters

Aslihan Ezgi Apaydin

Citation

Journal of Thoracic Oncology (2025) 20 (3): S208-S232. 10.1016/S1556-0864(25)00632-X

Authors

A.E.E. Apaydin1, M. Erman2, S.C. Onder1

Author affiliations

  • 1 Hacettepe University - Faculty of Medicine, Ankara/TR
  • 2 Hacettepe University, Ankara/TR

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 360P

Background

Programmed death-ligand 1 (PD-L1) expression level generally shows a positive relationship between response and survival but it has not proved useful as a predictive marker in second or further lines of therapy for NSCLC. LAG-3 and CD-155 inhibit antitumor immunity by interacting with various receptors and are targets for recently developed anti-LAG-3 and anti-T cell immunoreceptor with immunoglobulin and ITIM domain (TIGIT) agents. We investigated relationship between the expression levels of LAG-3 and CD-155 with overall survival (OS), progression-free survival (PFS), and overall response rates (ORR).

Methods

78 metastatic NSCLC patients who received nivolumab as second-line treatment after conventional chemotherapy were included. LAG-3 expression levels in tumor-infiltrating lymphocytes (TIL) and stromal lymphocytes (33 patients) and cytoplasmic and membranous CD-155 expression levels in tumor cells (61 patients) were evaluated by immunohistochemistry (IHC), and the relationship of the expressions with OS and PFS have been estimated with Kaplan-Meier method.

Results

Median OS of all patients was 8.7 months (95% CI: 5.1–12.3). We did not find any association between CD-155 and LAG-3 expressions and OS, PFS, and ORR. Median OS was 6.4 months (95% CI: 4.9–7.8) in patients with high CD-155 expression, and it was 11.3 months (95% CI: 6.9–15.8) in the low group (p=0,53). Positive LAG-3 expression in TIL was associated with a median OS of 6.8 months (95% CI: 5.1–8.6) while median OS was 6.9 months (95% CI: 2.3–11.5) in the negative group (p=0.8). Multivariate analyses identified poorer ECOG performance score, squamous histology, anemia, hypoalbuminemia, high pan-immune-inflammation value, and radiotherapy to the lung before nivolumab treatment as unfavorable independent risk factors for OS.

Conclusions

Our study could not establish the role of LAG-3 and CD-155 expressions in predicting the outcomes of nivolumab treatment. Both molecules have a key role in antitumor immunity mechanisms and further researches are needed.

Legal entity responsible for the study

The authors.

Funding

Hacettepe University Scientific Research Projects Coordination Unit.

Disclosure

M. Erman: Financial Interests, Personal and Institutional, Invited Speaker: Astellas, Deva, Abdi Ibrahim, Nobel, Roche, Bristol Myers Squibb, Merck Sharp & Dohme, Merck, Pfizer, Novartis, Eczacibasi; Financial Interests, Personal and Institutional, Funding: Roche, Merck, Sharp Dohme, Incyte, Bristol Myers Squibb, BeiGene, Novartis, Pfizer, Boehringer Ingelheim, AstraZeneca, Regeneron. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.