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Poster Display session

91P - Intracranial efficacy of crizotinib in advanced ROS1+NSCLC in real-world setting

Date

28 Mar 2025

Session

Poster Display session

Presenters

Zihua Zou

Citation

Journal of Thoracic Oncology (2025) 20 (3): S1-S97. 10.1016/S1556-0864(25)00632-X

Authors

Z. Zou1, G. Lin2, B. Cao3, D. Huang4, T. Lv5, M. Shi6, Y. Xia7, F. Ye8, P. Tian9, J. Hu10, Y. Fang11

Author affiliations

  • 1 Chinese Academy of Medical Sciences and Peking Union Medical College - National Cancer Center, Beijing/CN
  • 2 Fujian Cancer Hospital, 350014 - Fuzhou/CN
  • 3 Peking University Third Hospital, 100191 - Beijing/CN
  • 4 Tianjin Medical University Cancer Institute and Hospital, Tianjin/CN
  • 5 Jinling Hospital Affiliated to Nanjing University School of Medicine/Eastern Theater General Hospital of PLA, Nanjing/CN
  • 6 Jiangsu Cancer Hospital, Nanjing/CN
  • 7 The Second Affiliated Hospital of Zhejiang University School of Medicine, Hangzhou/CN
  • 8 The First Affiliated Hospital of Xiamen University, Xiamen/CN
  • 9 West China School of Medicine/West China Hospital of Sichuan University, Chengdu/CN
  • 10 Zhongshan Hospital Affiliated to Fudan University & Shanghai Geriatric Medical Center, Shanghai/CN
  • 11 Sir Run Run Run Shaw Hospital, Zhejiang University School of Medicine, Hangzhou/CN

Resources

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Abstract 91P

Background

There is limited real-world data about CNS efficacy of crizotinib in advanced ROS1+NSCLC.

Methods

Patients diagnosed with advanced ROS1+NSCLC with CNS metastases treated with crizotinib from multiple tertiary hospitals in China were retrospectively collected. Intracranial efficacy of crizotinib was evaluated by RECIST 1.1 (namely at most two target lesions (≥1 cm in MRI) could be chose). The definition of CNS progressive disease was that for patients with CNS target lesion, the sum of the largest diameter of target lesion was increased by at least 20% and 5 mm in number or new CNS lesion was reported; for patients without CNS target lesion, at least one non-target lesion enlarged significantly (at least increased by 5 mm) or new CNS lesion was found. CNS-PFS was calculated from the start date of crizotinib to the time of CNS progression.

Results

78 patients were included in this research. In patients with CNS target lesions (n=54), CNS-ORR was 66.7% and median CNS tumor shrinkage rate was 45% (0–100%). CNS-PFS was 22.2months (95%CI: 15.5–28.9m) in overall population. CNS-ORR (62.5% vs 78.6%, p=ns) and CNS-PFS (22.2 m vs 27.5 m, HR=1.16, p=0.687)was similar between patients who did not received brain-RT before or during the treatment of crizotinib and their counterparts. In patients with 1–5 CNS lesions, the addition of brain-RT did not prolong CNS-PFS (without RT: 26.6 mvs with RT: 27.5m, HR=1.24, p=0.65), for patients with >5 CNS lesions, similar outcomes were also reported (without RT: 15.9 m vs with RT: 21.1m, HR=1.44, p=0.58). The extra benefit by RT was also not confirmed in patients with larger CNS lesions (the sum of the largest diameter of CNS target lesions≥20 mm) (21.1 vs 18.8 m, HR=0.92, p=0.89), while CNS-PFS was numerically improved by RT in patients with smaller CNS lesions (the sum of the largest diameter of CNS target lesions

Conclusions

In contrast to the prior results, crizotinib demonstrated favourable CNS efficacy in ROS1+NSCLC even if brain-RT was not administered in our research. CNS-PFS was not significantly extended by RT in patients with multiple CNS metastases (>5) or larger CNS lesion (≥20 mm). A numerical improvement in CNS-PFS by RT was found in patients with smaller CNS lesion.

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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