Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster Display session

51P - Exploring the prognostic value of TP53 mutation in NSCLC in the era of precision oncology: A prospective real-world study

Date

22 Mar 2024

Session

Poster Display session

Topics

Tumour Site

Non-Small Cell Lung Cancer

Presenters

Matthew Kin Liang Chiu

Citation

Annals of Oncology (2024) 9 (suppl_3): 1-53. 10.1016/esmoop/esmoop102569

Authors

M.K.L. Chiu1, C.H.L. Wong1, J.K.S. Fong2, E.Y. Ko3, V.H.F. Lee1, T.Y. Kam4, K.M. Cheung5, S.F. Nyaw6, C.K. Kwan7, S.T.F. Mok8, A.W.M. Lee2, M.Y. Lim9

Author affiliations

  • 1 The University of Hong Kong - Li Ka Shing Faculty of Medicine, Hong Kong/HK
  • 2 The University of Hong Kong, Hong Kong/HK
  • 3 HKU - The University of Hong Kong, Hong Kong/HK
  • 4 Pamela Youde Nethersole Eastern Hospital, Hong Kong/HK
  • 5 Queen Elizabeth Hospital, Kowloon/HK
  • 6 Tuen Mun Hospital, New Territories/HK
  • 7 United Christian Hospital, Kowloon/HK
  • 8 Prince of Wales Hospital, New Territories/HK
  • 9 Princess Margaret Hospital, Kowloon/HK

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 51P

Background

TP53 is one of the most frequently mutated genes in non-small cell lung cancer (NSCLC) and has been associated with a worse prognosis. However, in the era of precision oncology, the prognostic value of TP53 co-mutation with other oncogenic genes remains controversial. Our study aimed to investigate the prognostic value of TP53 mutations in patients with advanced NSCLC.

Methods

Patients diagnosed with advanced NSCLC after March 2021 from seven public oncology centres in Hong Kong were enrolled and underwent FoundationOne next-generation sequencing prior to treatment. Relationship between TP53 mutation status and progression-free survival (PFS) or overall survival (OS) were analysed using survival analyses. Patients were further subdivided according to the presence of “druggable” or “non-druggable” co-mutations. The impact of using precision oncology-guided first-line treatment in the druggable population on survival was also assessed.

Results

A total of 426 patients with advanced NSCLC were included in this study. 244 (57%) harboured TP53 mutations (TP53 mt+) and 182 (42%) were TP53 wild-type (TP53WT). TP53 mt+ had a statistically significant worse PFS (hazard ratio (HR) 1.36; p=0.05) and OS (HR 1.47; p=0.04) when compared with TP53WT cohort. Furthermore, TP53mut+ with non-druggable targets had the worst survival outcomes, compared to TP53mut+ with druggable targets and TP53WT groups: 2-year PFS rate (18.3%, 35.1% and 37.8% respectively; p=0.033) and 3-year OS rate (12.5%, 66.7% and 57.1% respectively; p<0.001). Nevertheless, patients with TP53mt+ and druggable targets who received precision-oncology guided treatments had significantly better PFS (HR 0.50; p=0.01) and OS (HR 0.15; p<0.001) than those who did not. Patients who had EGFR or KRAS mutations receiving targeted first-line therapy had no significant difference in PFS or OS irrespective of TP53 status.

Conclusions

TP53mt+ imposes a negative impact on survival in patients with advanced NSCLC. However, this poor prognostic factor becomes negated in patients with druggable targets who receive targeted first-line therapy. Our study further highlights the importance of using precision-directed oncology treatment from the outset.

Legal entity responsible for the study

The University of Hong Kong.

Funding

Roche Hong Kong Limited.

Disclosure

All authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.