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Poster Display session

234P - Clinical findings and genomic characterization of NRG-1 alterations with a comprehensive genome profiling (CGP) in advanced non-small cell lung cancer (ANSCLC)

Date

22 Mar 2024

Session

Poster Display session

Topics

Pathology/Molecular Biology

Tumour Site

Non-Small Cell Lung Cancer

Presenters

Emanuele Vita

Citation

Annals of Oncology (2024) 9 (suppl_3): 1-6. 10.1016/esmoop/esmoop102579

Authors

E. Vita1, L. Mastrantoni2, A. Scala3, A. Vitale1, A. Stefani4, F. Monaca1, J. Russo1, G. Horn1, D. Occhipinti2, P. Troisi2, E. D'Argento2, A. Cosmai2, S. Polidori2, M. Di Salvatore5, A. Minucci1, C. nero3, G. Scambia3, G. Tortora2, E. Bria6

Author affiliations

  • 1 Fondazione Policlinico Universitario Agostino Gemelli IRCCS, Rome/IT
  • 2 Fondazione Policlinico Universitario Agostino Gemelli - IRCCS, Rome/IT
  • 3 Fondazione Policlinico Universitario Agostino Gemelli IRCCS, Rome/IT
  • 4 Fondazione Policlinico Universitario Agostino Gemelli - IRCCS Rome, Rome/IT
  • 5 Fondazione Policlinico Universitario Agostino Gemelli - IRCCS, 00168 - Rome/IT
  • 6 Fondazione Policlinico Universitario Agostino Gemelli IRCCS, Università Cattolica del Sacro Cuore, Roma/IT

Resources

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Abstract 234P

Background

Fusion events involving NRG-1, a member pan-HER pathway, have been highlighted as potential therapeutic target; however, little is known about epidemiological distribution and prognostic value of NRG alterations in large real-world datasets.

Methods

Data from from 592 patients with newly diagnosed ANSCLC undergone to CGP with DNA and RNA-based NGS (TSO500) from Jan 2020 to Oct 2023 were retrospectively collected in the context of the FPG500 program (interventional monocentric prospective study, NCT06020625).

Results

CGP analysis reported NRG-1 alterations in 58 samples (9.7%), including 32 deletions (5,4%), 23 sequence variants (3,8%), 1 NRG-fusion (0,1%), 2 amplifications (0,3%). NRG-1 alterations occurred together with other RTK-KRAS pathway events in 47 samples (81%) and/or with TP53 or BRCA/BRCA2 loss of function in 43 samples (74%). At the time of diagnosis, the median age was 68 y.o. Four pts (7%) had stage III disease and fifty-four (93%) pts had stage IV disease (median N° of metastatic sites = 1.5). Based on clinical evaluation, 40 pts (69%) were eligible for at least 1L treatment. The median 1L-PFS was 11.99 mo. (95%CI; 5.85 – 13.53) and the median OS was 20.26 mo. (95%CI: 11.30 – 24.60). Concurrent TP53 mutations did not resulted in statistically significant association (m1L-PFS p = 0.36; mOS: p = 0.57).

Conclusions

In our cohort, NRG-1 alterations resulted frequently associated with poor prognosis and main oncogene alterations and/or loss of function of oncosuppressor genes. Further investigations are needed.

Clinical trial identification

NCT06020625.

Legal entity responsible for the study

Fondazione Policlinico A Gemelli - IRCCS, Rome.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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