Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

E-Poster Display

170P - β2-adrenergic receptor gene expression as a prognostic and predictive biomarker in HER2-positive early-stage breast cancer patients enrolled on the NCCTG-N9831 (Alliance) trial

Date

17 Sep 2020

Session

E-Poster Display

Topics

Tumour Site

Breast Cancer

Presenters

Rafael Caparica

Citation

Annals of Oncology (2020) 31 (suppl_4): S303-S339. 10.1016/annonc/annonc267

Authors

R. Caparica1, Y. Ma2, C. De Angelis1, F. Richard3, C. Desmedt3, A.H. Awada1, M. Piccart4, E. Perez5, A. Moreno-Aspitia5, S. Badve6, E..A. Thompson2, E. de Azambuja1

Author affiliations

  • 1 Medical Oncology Department, Institute Jules Bordet, 1000 - Brussels/BE
  • 2 Division Of Biomedical Statistics And Informatics, Mayo Clinic, Jacksonville/US
  • 3 Laboratory For Translational Breast Cancer Research, Department Of Oncology, KU Leuven, Leuven/BE
  • 4 Medical Oncology, Institute Jules Bordet, 1000 - Brussels/BE
  • 5 Division Of Hematology And Oncology, Mayo CLinic, Jacksonville/US
  • 6 Department Of Pathology, Indiana University School of Medicine, Indiana/US

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 170P

Background

β2-adrenergic receptor (ADRB2) modulates immune activation, and may enhance the activity of trastuzumab in HER2-positive breast cancer cells. We assessed the impact of ADRB2 expression on the outcomes of patients enrolled on the NCCTG-N9831 trial.

Methods

NCCTG-N9831 compared chemotherapy (arm A) versus chemotherapy plus trastuzumab (arms B&C) as adjuvant treatment of HER2-positive early-stage breast cancer patients. Disease-free survival (DFS) was the primary endpoint. Gene expression data by DASL Assay were matched to patient’s characteristics. Median ADRB2 expression was assessed, with levels above median defined as ADRB2-high and below median as ADRB2-low. Stromal tumor-infiltrating lymphocytes (TILs) levels were correlated with ADRB2 expression. DFS curves were created by Kaplan Meier method; multivariate analyses were performed with Cox proportional hazard models.

Results

ADRB2 expression data were available for 1,282 patients (arm A [N=433]; arms B&C [N=849]), who were classified according to ADRB2 expression levels (ADRB2-high [N=944] and ADRB2-low [N=388]). Overall, ADRB2-high patients had a longer DFS than ADRB2-low patients (p=0.01). ADRB2-high was associated with a longer DFS in patients from arms B&C (p<0.01), but not in those from arm A (p=0.63). The addition of trastuzumab yielded a significant DFS improvement in ADRB2-high patients (DFS arms B&C versus arm A, p<0.01); whereas ADRB2-low patients did not benefit from trastuzumab (DFS arms B&C versus arm A, p=0.47). ADRB2 expression presented a direct correlation with TILs levels (r=0.24, p<0.001). High TILs levels were associated with a favorable prognosis in ADRB2-high patients (p=0.03), but not in ADRB2-low patients (p=0.12).

Conclusions

These results suggest that ADRB2 expression is associated with a favorable prognosis and could identify a subset of HER2-positive early-stage breast cancer patients who appear not to benefit from adjuvant trastuzumab.

Clinical trial identification

NCT00005970.

Editorial acknowledgement

Legal entity responsible for the study

North Central Cancer Treatment Group (NCCTG)-N9831 trial. NCCTG is now part of the Alliance for Clinical Trials in Oncology (Alliance).

Funding

Research reported in this publication was supported by the National Cancer Institute of the National Institutes of Health under Award Numbers U10CA180821, U10CA180882, and U24CA196171 (to the Alliance for Clinical Trials in Oncology). Genentech provided trastuzumab for this study. Also supported in part by funds from Genentech, Genomic Health, Breast Cancer Research Foundation; and Fondation Cancer Luxembourg (to F.R. and C.D.).

Disclosure

R. Caparica: Speaker Bureau/Expert testimony: Boehringer-Ingelheim; Speaker Bureau/Expert testimony, Travel/Accommodation/Expenses: AstraZeneca; Speaker Bureau/Expert testimony: Janssen; Travel/Accommodation/Expenses: Pfizer. A.H. Awada: Speaker Bureau/Expert testimony: Roche; Speaker Bureau/Expert testimony: Lilly; Speaker Bureau/Expert testimony: Amgen; Speaker Bureau/Expert testimony: ESAI; Speaker Bureau/Expert testimony: BMS; Speaker Bureau/Expert testimony: Pfizer; Speaker Bureau/Expert testimony: Novartis; Speaker Bureau/Expert testimony: MSD. M. Piccart: Honoraria (institution), Officer/Board of Directors: Radius; Honoraria (self), Honoraria (institution): AstraZeneca; Honoraria (self), Honoraria (institution): Lilly ; Honoraria (self), Honoraria (institution): MSD; Honoraria (self), Honoraria (institution): Novartis; Honoraria (self): Odonate; Honoraria (self), Honoraria (institution): Pfizer; Honoraria (self), Honoraria (institution): Roche; Honoraria (self): Crescendo biologics; Honoraria (self): Periphagen; Honoraria (self): Huya; Honoraria (self): Debiopharm; Honoraria (self): PharmaMar; Honoraria (self): G1 therapeutics; Honoraria (self): Menarini; Honoraria (self): Seattle Genetics; Honoraria (self): Immunomedics; Honoraria (institution): Synthon; Honoraria (institution): Servier. E. Perez: Honoraria (institution): NIH; Honoraria (institution): Roche. A. Moreno-Aspitia: Honoraria (institution): NIH; Honoraria (institution): Roche. S. Badve: Speaker Bureau/Expert testimony: Targos; Speaker Bureau/Expert testimony: MSD; Speaker Bureau/Expert testimony: Natera; Honoraria (institution): Dako/Agilent; Honoraria (institution): Roche/Ventana. E. de Azambuja: Honoraria (self), Honoraria (institution), Research grant/Funding (institution), Travel/Accommodation/Expenses: Roche; Travel/Accommodation/Expenses: Glaxo. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.