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E-Poster Display

601TiP - First-in-human phase I study of a novel oral Wee1 inhibitor (Debio 0123) in combination with carboplatin in patients with advanced solid tumours

Date

17 Sep 2020

Session

E-Poster Display

Topics

Clinical Research

Tumour Site

Presenters

Hans Gelderblom

Citation

Annals of Oncology (2020) 31 (suppl_4): S462-S504. 10.1016/annonc/annonc271

Authors

H. Gelderblom1, J.A. Gietema2, I.M.E. Desar3, N.M. Ajmone4, M. Jalving5, J.R. Kroep6, V. Nicolas7, A. Vaslin7, K. Tobal7, D. Purcea8, S.F. van Haren7, L. Damstrup7

Author affiliations

  • 1 Medical Oncology Dept, Leids Universitair Medisch Centrum (LUMC), 2333 ZA - Leiden/NL
  • 2 Medical Oncology, University Hospital Groningen (UMCG), 9700 RB - Groningen/NL
  • 3 Medical Oncology Dept., Radboud University Medical Centre Nijmegen Undo, 6500 HB - Nijmegen/NL
  • 4 Cardiology, Leids Universitair Medisch Centrum (LUMC), 2300 - Leiden/NL
  • 5 Medical Oncology Dept, University Hospital Groningen (UMCG), 9700 RB - Groningen/NL
  • 6 Medical Oncology Department, Leiden University Medical Center (LUMC), 2300 RC - Leiden/NL
  • 7 Clinical Research Department, Debiopharm International S.A., 1002 - Lausanne/CH
  • 8 Biostatistics, Debiopharm International SA, 1002 - Lausanne/CH

Resources

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Abstract 601TiP

Background

The Wee1 tyrosine kinase is activated upon DNA damage and regulates the G2-M cell cycle checkpoint. Inhibition of Wee1, in conjunction with additional genetic alterations and/or addition of a DNA damaging agent, results in mitotic catastrophe and apoptosis of cancer cells, being an attractive approach for treating cancer. Debio 0123 is a potent and highly specific WEE1 inhibitor with an IC50 in the low nanomolar range. Debio 0123 was demonstrated to inhibit phospho-CDC2 which translated into an increase in DNA damage and premature entry into mitosis (AACR 2019, abstract 4423). Debio 0123 combination with carboplatin (CbPt) was synergic in vitro. In vivo, Debio 0123 was demonstrated to increase antitumoral activity of cbPt in models where neither agent was active alone.

Trial design

Methods This is a phase I, multi-center, open-label, dose escalation study of Debio 0123 as monotherapy (first cycle only) and in combination with CbPt, from cycle 2 in subjects with advanced solid tumors that recurred or progressed following prior platinum therapy. Primary objective: determination of the recommended phase II dose (RP2D) of Debio 0123 when administered in combination with CbPt using a modified Continual Reassessment Method (mCRM). Patients receive Debio 0123 orally once daily for the first 3 days of a 21 day-cycle as monotherapy during first cycle and in combination with CbPt in following cycles. Secondary objectives: includes determination of occurrence of dose-limiting toxicities (DLT) and characterization of the pharmacokinetics of Debio 0123 and its active metabolite, which are evaluated after single and repeated administration when administered alone or in combination with CbPt. Potential risk of QTc prolongation is evaluated by exposure-response modeling. Pharmacodynamics biomarkers including phospho-CDC2 are explored in pre- and post-treatment tumors and skin biopsies. Recruitment started in July 2019. Cohort 2 has been completed. Start of enrollment into cohort 3 is currently unknow due to COVID-19 but will begin as soon as possible.

Clinical trial identification

NCT03968653.

Editorial acknowledgement

Legal entity responsible for the study

Debiopharm Interbational S.A.

Funding

Debiopharm International S.A.

Disclosure

V. Nicolas: Full/Part-time employment: Debiopharm AS. A. Vaslin: Full/Part-time employment: Debiopharm AS. K. Tobal: Full/Part-time employment: Debiopharm AS. D. Purcea: Full/Part-time employment: Debiopharm AS. S.F. van Haren: Full/Part-time employment: Debiopharm AS. L. Damstrup: Full/Part-time employment: Debiopharm International S.A.. All other authors have declared no conflicts of interest.

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