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E-Poster Display

647P - Clinical implications of intraductal carcinoma of the prostate sub-patterns in metastatic castration-resistant prostate cancer patients treated with abiraterone or docetaxel as the first-line therapy

Date

17 Sep 2020

Session

E-Poster Display

Topics

Tumour Site

Prostate Cancer

Presenters

Wang Zhipeng

Citation

Annals of Oncology (2020) 31 (suppl_4): S507-S549. 10.1016/annonc/annonc275

Authors

W. Zhipeng1, S. Zhu2, J. Chen3, J. Zhao2, X. Zhang4, G. Sun2, N. Chen2, H. Zeng5

Author affiliations

  • 1 Urology, West China Hospital, Sichuan University, 610041 - Chengdu/CN
  • 2 Urology, West China Hospital of Sichuan University, 610041 - Chengdu/CN
  • 3 Urology, WCSM/WCH - West China School of Medicine/West China Hospital of Sichuan University, 610041 - Chengdu/CN
  • 4 Urology, WCSM/WCH - West China School of Medicine/West China Hospital of Sichuan University, 610041 - chengdu/CN
  • 5 Department Of Urology, West China Hospital, Sichuan University, 610041 - Chengdu/CN

Resources

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Abstract 647P

Background

Intraductal carcinoma of the prostate (IDC-P) was separated into 2 patterns: pattern 1-loose cribriform or micropapillary; pattern 2-solid or dense cribriform. While the role of different IDC-P patterns remains obscure. The purpose of this study is to explore whether two distinct IDC-P patterns respond differently to abi and doc for metastatic castration-resistant prostate cancer (mCRPC) patients. The secondary goal was to compare the therapeutic efficacy between doc and abi for patients with different IDC-P patterns.

Methods

197 mCRPC patients were enrolled in this study. IDC-P was separated into pattern 1 and pattern 2. Patients received abiraterone (abi) or docetaxel (doc) as first-line treatment. Kaplan-Meier curves and Cox regression were used for survival analyses. The primary endpoint was PSA-progression free survival (PSA-PFS) and the secondary endpoints were overall survival (OS) and PSA response.

Results

In the cohort treated with abi as the first-line therapy, patients with IDC-P pattern 2 had obviously shorter PSA-PFS than IDC-P (-) patients (P=0.026) and patients with IDC-P pattern 1 ( P=0.038). In the cohort treated with doc as the first-line therapy, IDC-P (+) patients had poorer PSA-PFS than IDC-P (-) patients (P=0.020). IDC-P pattern 2 were associated with worse PSA-PFS than IDC-P (-) (P=0.004). No significant difference in PSA-PFS and PSA response was found between abi and doc in those IDC-P (-) patients. However in those IDC-P (+) patients, abi could significantly extend PSA-PFS (P=0.001) and improved PSA response (P=0.032) compared to doc. PSA-PFS was significantly shorter in the doc group than in the abi group, both in IDC-P pattern 1 (P=0.003) and IDC-P pattern 2 patients (P=0.003).

Conclusions

This study demonstrated that two different architectural patterns of IDC-P among mCRPC patients might impact disease progression. IDC-P pattern 2 was associated with faster disease progression. Abiraterone instead of docetaxel might be more suitable in mCRPC patients with IDC-P, whether it is in IDC-P pattern 1 or IDC-P pattern 2.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

Hao Zeng.

Funding

National Natural Science Foundation of China.

Disclosure

All authors have declared no conflicts of interest.

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