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E-Poster Display

265P - Circulating PIK3CA mutation detection at diagnosis in non-metastatic inflammatory breast cancer patients

Date

17 Sep 2020

Session

E-Poster Display

Topics

Tumour Site

Breast Cancer

Presenters

Violette Allouchery

Citation

Annals of Oncology (2020) 31 (suppl_4): S340-S347. 10.1016/annonc/annonc260

Authors

V. Allouchery1, A. Perdrix2, C. Calbrix2, A. Berghian3, J. Lequesne4, M. Fontanilles5, M. Leheurteur5, P. Etancelin2, N. Sarafan Vasseur6, F. Di Fiore7, F. Clatot5

Author affiliations

  • 1 Department Of Medical Oncology, Centre Henri Becquerel, 76000 - Rouen/FR
  • 2 Department Of Bio-pathology, Centre Henri Becquerel, 76000 - Rouen/FR
  • 3 Department Of Pathology, Centre Henri Becquerel, 76000 - Rouen/FR
  • 4 Clinical Research Unit, Centre Henri Becquerel, 76000 - Rouen/FR
  • 5 Department Of Medical Oncology, Centre Henri Becquerel, 76038 - Rouen/FR
  • 6 Normandie University, Inserm 1245, Rouen university Hospital, 76031 - Rouen/FR
  • 7 Department Of Medical Oncology, Centre Henri Becquerel, 76000 - rouen/FR

Resources

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Abstract 265P

Background

Inflammatory breast cancer (IBC) is an aggressive BC subtype with rapid onset and poor outcomes. After biopsy confirmation, first-line treatment is based on systemic chemotherapy. A targetable somatic PIK3CA mutation is reported in 30% of IBC, allowing for treatment by PI3Kα-specific inhibitors, such as alpelisib. The aim of this study was to evaluate the detection rate of circulating PIK3CA mutation in non-metastatic IBC patients harbouring a PIK3CA mutation on initial biopsy.

Methods

This monocentric retrospective study was based on available stored plasmas and biopsies at diagnosis and included all non-metastatic IBC patients who were treated by neo-adjuvant chemotherapy. PIK3CA mutations (E542K, E545K, H1047R/L) were assessed by droplet digital PCR (ddPCR) in plasma samples and tumoral tissue at diagnosis.

Results

A total of 54 patients treated between 2008 and 2018 in Henri Becquerel Center and with sufficient DNA from tumor tissue were included. 14/54 patients (26%) had a PIK3CA mutation identified on baseline biopsy (H1047R=8; H1047L=3; E545K=2; E542K=1). All mutations were single. Among them, 11 (76%) patients had enough circulating DNA for circulating mutations analyses. Corresponding circulating PIK3CA mutations were found in 4/11 patients (36%). Beside these 4 patients, no other circulating mutation was identified among the 42 patients with fully interpretable circulating and biopsy mutational analyses.

Conclusions

Our results showed that corresponding circulating PIK3CA mutation was identified in 36% of non-metastatic IBC patients with baseline somatic PIK3CA mutation on tumor tissue while no circulating mutation was found among non-mutated PIK3CA patients. Further studies are needed to determine the prognostic and therapeutic impact of circulating PIK3CA mutation in IBC patients.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

Henri Becquerel Center.

Funding

Henri Becquerel Center La Ligue Contre le Cancer.

Disclosure

M. Fontanilles: Travel/Accommodation/Expenses: F. Hoffmann–La Roche; Travel/Accommodation/Expenses: GSK. F. Di Fiore: Honoraria (self): Roche; Honoraria (self): Amgen; Honoraria (self): Merck; Honoraria (self): Sanofi; Honoraria (self): Bayer; Honoraria (self): Servier; Honoraria (self): Janssen. F. Clatot: Honoraria (self), Travel/Accommodation/Expenses: BMS; Honoraria (self), Travel/Accommodation/Expenses: Lilly; Honoraria (self), Travel/Accommodation/Expenses: Merck; Honoraria (self), Research grant/Funding (institution), Travel/Accommodation/Expenses: Roche; Research grant/Funding (institution): AstraZeneca. All other authors have declared no conflicts of interest.

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