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E-Poster Display

113P - C-reactive protein (CRP) levels in immune checkpoint inhibitor response and progression in advanced non-small cell lung cancer: A bi-center study

Date

17 Sep 2020

Session

E-Poster Display

Topics

Translational Research

Tumour Site

Thoracic Malignancies

Presenters

Florian Posch

Citation

Annals of Oncology (2020) 31 (suppl_4): S274-S302. 10.1016/annonc/annonc266

Authors

F. Posch1, D. Barth1, W.M. Brueckl2, G. Zeitler2, V. Foris3, S. Mollnar1, M. Stotz1, C.H. Rossmann1, A. Terbuch1, M. Balic1, T. Niedrist4, T. Bertsch5, H. Stöger1, M. Pichler1, H. Olschewski3, G. Absenger1, J.H. Ficker2, A. Gerger1, J.M. Riedl1

Author affiliations

  • 1 Division Of Oncology; Department Of Internal Medicine, Medical University of Graz, 8036 - Graz/AT
  • 2 Department Of Respiratory Medicine, Allergology And Sleep Medicine / Nuremberg Lung Cancer Center,, Nuremberg General Hospital, 90419 - Nürnberg/DE
  • 3 Division Of Pulmonology; Department Of Internal Medicine, Medical University of Graz, 8036 - Graz/AT
  • 4 Clinical Institute Of Medical And Chemical Laboratory Diagnostics, Medical University of Graz, 8036 - Graz/AT
  • 5 Institute Of Laboratory Medicine; Nuremberg General Hospital, Nuremberg General Hospital, 90419 - Nürnberg/DE

Resources

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Abstract 113P

Background

Biomarkers for predicting response to immune checkpoint inhibitors (ICI) are scarce and often lack external validation. This retrospective bi-center study provides a comprehensive investigation of pretreatment C-reactive protein (CRP) levels and its longitudinal trajectories as a marker of treatment response and disease outcome in patients with advanced non-small cell lung cancer (NSCLC) undergoing immunotherapy with anti PD-1 or anti PD-L1 agents.

Methods

Primary treatment outcomes were progression-free survival (PFS) and overall survival (OS), while the objective response rate (ORR) was a secondary outcome, respectively. Joint models were implemented to evaluate the association of longitudinal CRP trajectories and progression risk.

Results

In the discovery cohort (n=90), elevated pre-treatment CRP emerged as independent predictor of worse PFS (HR per doubling of baseline CRP=1.37, 95%CI: 1.16-1.63, p<0.0001), worse OS (HR per doubling of baseline CRP=1.42, 95%CI: 1.18-1.71, p<0.0001) and a lower ORR ((Odds Ratio (OR) of ORR per doubling of baseline CRP=0.68, 95%CI: 0.51-0.92, p=0.013)). In the validation cohort (n=101), pre-treatment CRP could be confirmed as a predictor of PFS and OS, but not ORR. Elevated trajectories of CRP during anti PD-(L)1 treatment (Adjusted HR per 10mg/L increase in CRP=1.22, 95%CI: 1.15-1.30, p<0.0001), as well as a faster increases of CRP over time (HR per 10mg/L/month faster increase in CRP levels =13.26, 95%CI: 1.14-154.54, p=0.039) were strong predictors of an elevated progression risk, whereas an early decline of CRP was significantly associated with a reduction in PFS risk (HR=0.91, 95%CI: 0.83-0.99, p=0.036), respectively.

Conclusions

These findings warrant that CRP should be further explored by prospective studies as a non-invasive biomarker for assessing treatment benefit during anti PD-(L)1 treatment in advanced NSCLC.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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