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E-Poster Display

104P - Angiogenesis-related factors and clinical outcomes in combination therapy with paclitaxel (PTX), ramucirumab (RAM) plus nivolumab (Nivo) for advanced gastric cancer (AGC)

Date

17 Sep 2020

Session

E-Poster Display

Topics

Immunotherapy;  Translational Research

Tumour Site

Gastric Cancer

Presenters

Naoki Izawa

Citation

Annals of Oncology (2020) 31 (suppl_4): S274-S302. 10.1016/annonc/annonc266

Authors

N. Izawa1, S. Ohta2, S. Kadowaki3, T. Kajiwara4, K. Minashi5, Y. Sunakawa1, K. Muro3, T. Nishina4, S. Hironaka6, T. Yamanaka7, T.E. Nakajima8, Y. Kawakami9

Author affiliations

  • 1 Department Of Clinical Oncology, St. Marianna University School of Medicine, 216-8511 - Kawasaki/JP
  • 2 Division Of Cellular Signaling Institute For Advanced Medical Research, Keio University School of Medicine, Tokyo/JP
  • 3 Department Of Clinical Oncology, Aichi Cancer Center Hospital, 464-8681 - Nagoya/JP
  • 4 Department Of Gastrointestinal Medical Oncology, National Hospital Organization Shikoku Cancer Center, 791-0280 - Matsuyama/JP
  • 5 Clinical Trial Promotion Dept, Chiba Cancer Center, Chiba/JP
  • 6 Department Of Medical Oncology And Hematology, Oita University Faculty of Medicine, 879-5593 - Yufu/JP
  • 7 Biostatistics Department, Yokohama City University Hospital, 236-004 - Yokohama/JP
  • 8 Kyoto Innovation Center For Next Generation Clinical Trials And Ips Cell Therapy, Kyoto University Hospital, 606-8507 - Kyoto/JP
  • 9 School Of Medicine International University Of Health And Welfare, Department of Immunology, 286-0048 - Narita/JP

Resources

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Abstract 104P

Background

Previous studies showed that angiogenesis-related factors could play a key role in the development of immunosuppressive microenvironment and may serve as predictors for chemotherapy. This biomarker study was focused on the dynamics of angiogenesis-related factors in clinical trial of combination therapy with PTX, RAM plus Nivo for AGC as 2nd-line treatment, with expected antitumor activity [Kadowaki S, et al, Ann Oncol, 2019 Jul;30 Suppl4].

Methods

We evaluated plasma concentrations of angiogenesis-related factors in plasma at baseline, 4 weeks after treatment, and progression and the association with clinical outcomes in patients with available samples. PlGF, VEGF-A/D, HGF, IL-6/8, angiopoietin-2, sNeurophilin-1, sICAM-1, sVCAM-1, sVEGFR-1/2/3, and INF-γ were measured by magnetic bead panel Milliplex xMAP kits, Flt3L and sPD-L1 by ELISA. Progression-free survival (PFS) and overall survival (OS) were estimated using the Kaplan-Meier method and compared by the log-rank test. Fisher’s exact test was used to compare IL-6 level and response.

Results

Forty-one of 43 AGC pts were enrolled: median age, 66 years; male, 82.9%; ECOG PS 1, 48.8%; and diffuse type, 43.9%. Plasma average levels of PlGF, VEGF-A/D, and sVCAM-1 significantly increased after treatment; whereas angiopoietin-2 exhibited a decrease during treatment. When the median value was adopted as cut-off value, high levels of IL-8 and sICAM-1 at baseline were significantly associated with shorter OS [HR 2.69 (95%CI 1.28-5.92), p = 0.009; HR 3.50 (95%CI 1.63-8.16), p = 0.001, respectively]; whereas, comparable PFS. The elevation of IL-6 level at 4 weeks were detected in 8 (35%) of 23 responders and in only 2 (15%) of 13 non-responders (P = 0.21). Moreover, it was significantly associated with favorable OS [HR 0.20 (95%CI 0.05-0.60), p = 0.002], but not PFS.

Conclusions

This study identified baseline plasma levels of IL-8 and sVCAM-1 as potential prognostic markers corresponding to favorable OS for combination therapy of PTX, RAM plus Nivo in patients with AGC. In addition, the increase in IL-6 level at 4 weeks may predict clinical benefit from the combination therapy.

Clinical trial identification

UMIN000025947.

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Ono pharmaceutical company.

Disclosure

N. Izawa: Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd ; Research grant/Funding (institution): Eli Lilly Japan. S. Ohta: Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd . S. Kadowaki: Honoraria (self), Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd; Honoraria (self), Research grant/Funding (institution): Eli Lilly Japan. K. Minashi: Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd. Y. Sunakawa: Honoraria (self), Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd; Honoraria (self), Research grant/Funding (institution): Eli Lilly Japan. K. Muro: Honoraria (self), Advisory/Consultancy, Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd ; Honoraria (self), Advisory/Consultancy, Travel/Accommodation/Expenses: Eli Lilly Japan; Advisory/Consultancy: Nihon Kayaku. T. Nishina: Honoraria (self), Advisory/Consultancy, Research grant/Funding (self): ONO Pharmaceutical CO.,Ltd; Honoraria (self), Advisory/Consultancy, Research grant/Funding (self): Eli Lilly Japan; Honoraria (self): Nihon kayaku. S. Hironaka: Honoraria (self), Advisory/Consultancy, Research grant/Funding (institution): Ono Pharmaceutical Co., Ltd. T.E. Nakajima: Honoraria (self), Research grant/Funding (self), Research grant/Funding (institution): Ono Pharmaceutical Co., LTD ; Honoraria (self), Research grant/Funding (self), Research grant/Funding (institution): Eli Lilly Japan; Honoraria (self), Research grant/Funding (self), Research grant/Funding (institution): Nihon kayaku. Y. Kawakami: Honoraria (self), Research grant/Funding (self): Ono Pharmaceutical Co., Ltd. All other authors have declared no conflicts of interest.

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