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E-Poster Display

602TiP - A first-in-human study of NOV1601 (CHC2014), small-molecule inhibitor of TRK family proteins, in adult patients with solid organ malignancies

Date

17 Sep 2020

Session

E-Poster Display

Topics

Clinical Research

Tumour Site

Presenters

Tak Yun

Citation

Annals of Oncology (2020) 31 (suppl_4): S462-S504. 10.1016/annonc/annonc271

Authors

T. Yun1, Y.W. Moon2, S.J. Shin3, D. Kim4, C.H. Yun5, J.Y. You6, B. Hyun7, H.W. Cho8, J.S. Kim8, G. Im9, Y. Kim9, N. Park9

Author affiliations

  • 1 Center For Specific Organs Cancer, National Cancer Center, 10408 - Goyang-si, Gyeonggi-do/KR
  • 2 Department Of Internal Medicine, CHA Bundang Medical Center, Seongnam-si, Gyeonggi-do/KR
  • 3 Medical Oncology, Severance Hospital, Yonsei University Health System, Seoul/KR
  • 4 Department Of Internal Medicine, Seoul National University Hospital, Seoul/KR
  • 5 New Drug & Bio Research Center, HANDOK Inc., Seongnam-si, Gyeonggi-do/KR
  • 6 Clinical Research Science, HANDOK Inc., Seoul/KR
  • 7 Clinical Research Operation, HANDOK Inc., Seoul/KR
  • 8 New Drug Discovery Center, CMG Pharmaceutical Inc., Seongnam-si, Gyeonggi-do/KR
  • 9 Clinical Development, National OncoVenture, Goyang-si, Gyeonggi-do/KR

Resources

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Abstract 602TiP

Background

TRK family proteins (Trk A, B and C) are constitutively activated in certain malignancies of both children and adults, especially when chimeric proteins involving tyrosine kinase portion are produced by rearrangements of NTRK 1, 2 or 3 genes. A number of small molecule inhibitors of TRK proteins have been developed and tried in clinical trials, some of which are now approved in an accelerated pathway. Cases of primary or secondary resistance to such drugs are accumulating, so a new drug with broader activity and improved pharmacologic features is still warranted. NOV1601 (CHC2014) is a novel small molecule of oral route of administration, with unique dynamic configurations in relation to solvent-front mutants of TRK proteins. Non-clinical studies suggested potential of NOV1601 (CHC2014) to overcome primary or secondary resistance to the already approved TRK inhibitor.

Trial design

This is the first-in-human, phase I, open-label, multicenter, dose-escalation study to investigate the safety, tolerability, PK, and clinical activity of NOV1601 in subjects with solid organ malignancies at four research-based hospitals in the Republic of Korea. The primary objective is to determine the recommended phase II dose (RP2D) of NOV1601 in adult subjects with solid organ malignancies. Dose escalation will follow a 3+3 design and will be based on prior cohort review. NOV1601 will be administered orally once daily (QD) or twice daily (BID) doses for continuous 28-day cycles. To date, 7 patients were enrolled and administered at 50mg QD or 100mg QD. NOV1601 is currently being administered at 150mg QD. The maximum tolerated dose (MTD) is not pre-determined yet. If MTD is not reached because of no dose-limiting toxicity, then the RP2D will be determined by PK and safety profiles. Efficacy per RECIST convention will be measured as an exploratory endpoint, and special attention will be cast to subjects with known genetic alterations involving NTRK family genes. Such cases will be validated at a central pathology laboratory using multiple diagnostic technologies available. Currently, this study is ongoing with affirmative PK and safety profiles as had been expected from non-clinical studies.

Clinical trial identification

NCT04014257.

Editorial acknowledgement

Legal entity responsible for the study

HANDOK INC. & CMG Pharmaceutical Inc. & National OncoVenture.

Funding

HANDOK INC. & CMG Pharmaceutical Inc. & National OncoVenture.

Disclosure

All authors have declared no conflicts of interest.

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