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Poster Display & Cocktail

23P - Anti-tumor activity of novel Globo-H targeting CAR T cells in cholangiocarcinoma model

Date

03 Mar 2025

Session

Poster Display & Cocktail

Presenters

Francesca Putti

Citation

Annals of Oncology (2025) 10 (suppl_2): 1-2. 10.1016/esmoop/esmoop104178

Authors

F. Putti1, L. Roz2, F. Murianni2, G. Bertolini2, M. Gentili2, F. Zonca1, M. Figini3, E. Luison3, F.G.M. De Braud4, M. Di Nicola5, F. De Santis1

Author affiliations

  • 1 Oncologia Medica 1, Fondazione IRCCS - Istituto Nazionale dei Tumori, 20133 - Milan/IT
  • 2 Genomica Tumorale, Fondazione IRCCS - Istituto Nazionale dei Tumori, 20133 - Milan/IT
  • 3 Anatomia Patologica 2, Fondazione IRCCS - Istituto Nazionale dei Tumori, 20133 - Milan/IT
  • 4 Medical Oncology & Haemathology Department, Fondazione IRCCS - Istituto Nazionale dei Tumori, 20133 - Milan/IT
  • 5 Medical Oncology And Hematology, Fondazione IRCCS - Istituto Nazionale dei Tumori, 20133 - Milan/IT

Resources

This content is available to ESMO members and event participants.

Abstract 23P

Background

Chimeric Antigen Receptor (CAR) T-cell therapy has been a clinical breakthrough in hematological malignancies; however, it is still facing multifactorial challenges for clinical success in solid tumor, such as the selection of a proper target to avoid on-target off-tumor toxicities. To this extent, we characterized the monoclonal antibody MBr1 that specifically recognizes Globo-H cell-surface glycan to develop novel CAR T cells. Globo-H is overexpressed on several epithelial solid cancers, including the treatment-orphan cholangiocarcinoma, while it is minimally expressed on normal tissues, making it an attractive tumor-associated antigen.

Methods

MBr1 specific single chain variable fragment (scFv) was cloned into second generation retroviral vectors to generate both anti-Globo-H.CAR.CD28 and anti-GloboH.CAR.41BB T cells. Then, they were ex-vivo expanded, phenotypically characterized and assessed preclinically for their anti-tumor responses.

Results

We demonstrated that anti-Globo-H.CAR T cells exert robust anti-tumor activity mediated by Globo-H specific induction in vitro and in vivo. Indeed, both anti-Globo-H.CAR.CD28 and anti-GloboH.CAR.41BB T cells specifically killed Globo-H-positive cancer cell lines, whereas Globo H-negative tumor cells were not targeted. Although we have not found differences in terms of anti-tumor activity between anti-GloboH.CAR.41BB and anti-Globo-H.CAR.CD28 T cells, they showed significantly distinct phenotypic traits. The first displayed higher frequency of early differentiated T cells (Central Memory, TCM and Stem cell memory, TSCM), while the latter was enriched in effector memory T cells (TEM), conferring higher persistency or rapid effector functions, respectively. Lastly, we infused anti-Globo-H.CAR T cells in EGI-1 (cholangiocarcinoma cell line) xenotransplanted NSG mice, resulting in a significant reduction of the tumor volume following their homing in the tumor bed.

Conclusions

Our data strongly validated the antigen-mediated cytotoxicity of anti-Globo-H.CAR T cells in vitro, and their anti-tumor efficacy in vivo. Experiments conducted under GMP procedures are ongoing to validate its therapeutic potential paving the way for its clinical translation.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Regione Lombardia: FORCE4CURE project.

Disclosure

All authors have declared no conflicts of interest.

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