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Poster Display & Cocktail

68P - Adjuvant effect of maraviroc in combination with cisplatin on tumor growth and survival in a murine model of gastric cancer

Date

03 Mar 2025

Session

Poster Display & Cocktail

Presenters

Priscilla Brebi

Citation

Annals of Oncology (2025) 10 (suppl_2): 1-8. 10.1016/esmoop/esmoop104219

Authors

P. Brebi1, B. Mora2, M. Reyes2, L. Lobos3, C. Ili1, Y. Mora1, K. Buchegger1, T. Millapan1

Author affiliations

  • 1 Laboratory Of Integrative Biology (libi), Universidad de La Frontera, 4810296 - Temuco/CL
  • 2 Institute Of Biomedical Sciences, Universidad Autónoma de Chile, 4780000 - Temuco/CL
  • 3 Centro De Medicina Regenerativa, Facultad de Medicina Clinica Alemana Universidad del Desarrollo, 7590943 - Las Condes/CL

Resources

This content is available to ESMO members and event participants.

Abstract 68P

Background

Chemoresistance remains a significant challenge in the treatment of gastric cancer, leading to poor prognosis and limited therapeutic options. The CCL5/CCR5 axis has been implicated in tumor progression and resistance to chemotherapeutics. Given the critical role of this axis in cancer progression, this study evaluated the potential of Maraviroc (MVC), a CCR5 antagonist, as an adjuvant to cisplatin (CDDP) to overcome chemoresistance in a murine model of gastric cancer.

Methods

A murine xenograft model using gastric tumor cells was employed. Tumoroids were formed from these cells and injected subcutaneously into immunocompromised BALB/c NOD/SCID mice. Animals were then randomized into four groups: a control group treated with saline, a group treated with maraviroc (MVC), a group treated with cisplatin (CDDP), and a group treated with a combination of MVC and CDDP. Tumor growth and survival were monitored for 21 days. Biochemical analyses were performed to assess the metabolic profile of the animals.

Results

The combination of MVC and CDDP did not induce a greater reduction in tumor volume compared to CDDP alone. However, the co-administration of MVC significantly improved the survival of treated animals (Figure 1). The combination MVC/CDDP attenuated the adverse effects of CDDP on biochemical parameters such as glucose, lactate, and creatinine levels.

Conclusions

The results suggest that MVC could act as a sensitizing agent, enhancing the efficacy of CDDP while reducing its toxicity. Although the exact mechanism of this synergy is not yet fully elucidated, it is hypothesized that blocking CCR5 could modulate the tumor immune response and reduce resistance to chemotherapy. These findings open new avenues for the development of combined therapeutic strategies for the treatment of gastric cancer. Future studies should delve deeper into the underlying molecular mechanisms and evaluate the efficacy of this combination in more complex preclinical models.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Agencia Nacional de Investigación y Desarrollo (ANID).

Disclosure

All authors have declared no conflicts of interest.

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