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Poster Display

19P - Effects of CTLA-4 Single Nucleotide Polymorphisms (SNPs) on toxicity of ipilimumab-containing regimens in patients with advanced stage melanoma

Date

08 Dec 2022

Session

Poster Display

Presenters

Karlijn Joode

Citation

Annals of Oncology (2022) 16 (suppl_1): 100100-100100. 10.1016/iotech/iotech100100

Authors

K.D. Joode1, R. Van Schaik2, A. Zippelius3, A.A.M. Van der Veldt4, C.L. Gerard5, H. Läubli6, O.A. Michielin7, R.A.F. von Moos8, M. Joerger9, M.P. Levesque10, S. Aeppli11, J. Mangana10, C. Mangas12, S. Meyer13, S. Leoni-Parvex14, R.H. Mathijssen15, Y. Metaxas16

Author affiliations

  • 1 Erasmus MC Cancer Institute, Erasmus University Medical Center, Rotterdam/NL
  • 2 Erasmus University Medical Center, Rotterdam/NL
  • 3 University Hospital of Basel, Basel/CH
  • 4 Erasmus MC - University Medical Center, Rotterdam/NL
  • 5 Lausanne University Hospital (CHUV), Lausanne/CH
  • 6 University Hospital and University of Basel, Basel/CH
  • 7 Centre Hospitalier Universitaire Vaudois - CHUV, Lausanne/CH
  • 8 KSGR - Kantonsspital Graubünden, Chur/CH
  • 9 Kantonsspital St. Gallen, St. Gallen/CH
  • 10 University Hospital Zurich, Zurich/CH
  • 11 Cantonal Hospital St. Gallen, St. Gallen/CH
  • 12 EOC - Ospedale Regionale Bellinzona e Valli - Istituto Oncologico della Svizzera Italiana (IOSI), Bellinzona/CH
  • 13 Blood Transfusion Service Zürich, Swiss Red Cross, Schlieren/CH
  • 14 Istituto Cantonale di Patologia, Locarno/CH
  • 15 Medical Oncology Department, Erasmus MC Cancer Institute, Erasmus University Medical Center, 3075 EA - Rotterdam/NL
  • 16 Spital Thurgau AG - Kantonsspital Muensterlingen, Muensterlingen/CH

Resources

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Abstract 19P

Background

SNPs of the cytotoxic T-lymphocyte-associated protein 4 (CTLA-4) gene, an inhibitor of T cell priming, are associated with auto- and allo-immunity. Previously, studies implied a role for these SNPs as surrogate markers for outcome in melanoma patients treated with immune checkpoint inhibitors. However, no predictive SNPs are defined to date. The primary aim of this study was to analyze different CTLA-4 SNPs in a large real-world cohort of melanoma patients treated with ipilimumab and to correlate these SNPs with toxicity and survival.

Methods

Archival blood and/or tumor-tissue samples were collected from 317 advanced stage melanoma patients treated with ipilimumab (+/- nivolumab) in 5 Swiss and Dutch hospitals. DNA was extracted and used for MALDI-TOF MS based genotyping for 11 different SNPs. Associations between different allele genotypes and occurrence of grade ≥3 toxicity, treatment response and survival outcomes were tested using univariable logistic regressions or Cox proportional hazard models.

Results

DNA of 216/317 patients could be analyzed. The cohort was representative of a real-life population of metastatic melanoma patients receiving ipilimumab (+/- nivolumab): among the remaining 216/317 evaluable patients, 65.3% were male, median age at diagnosis was 59 years, 37% of patients had partial or complete response and 59% had ≥1 immune-related adverse events. A TT-genotype in the -1722 C/T SNP (rs733618, T=0.8345 (1000Genomes)), located in the promoter region of CTLA-4, was significantly associated with a decreased rate of ≥ grade 3 toxicity [odds ratio=0.40; CI95=0.16 - 1.00; p=0.049]. The TT-genotype in the Jo27 T/C SNP (rs11571297, T=0.5575 (1000Genomes)) [hazard ratio (HR)=0.54; CI95=0.28-1.02, p=0.056] and the GG-genotype in the Jo31 SNP (rs11571302, G=0.5713 (1000Genomes) [HR=0.54; CI95 = 0.29-0.99, p=0.046], both located at the 3’ untranslated region, were associated with increased overall survival.

Conclusions

CTLA-4 SNPs might be predictive of toxicity and/or survival in melanoma patients receiving ipilimumab. Confirmatory studies are needed to exploit findings of this exploratory study and to investigate the exact role of CTLA-4 SNPs as possible biomarkers.

Legal entity responsible for the study

The authors.

Funding

BMS.

Disclosure

K.D. Joode: Financial Interests, Personal, Other, Travel expenses congress: Ipsen. A.A.M. Van der Veldt: Financial Interests, Institutional, Advisory Board: BMS, MSD, Merck, Pfizer, Ipsen, Eisai, Pierre Fabre, Roche, Novartis, Sanofi. R.H. Mathijssen: Financial Interests, Personal, Invited Speaker: Novartis; Financial Interests, Personal, Advisory Role: Servier; Financial Interests, Personal, Licensing Fees, Patent pending: Pamgene; Financial Interests, Institutional, Research Grant, Investigator-initiated research: Astellas, Bayer, Boehringer Ingelheim, Cristal Therapeutics, Pamgene, Pfizer, Novartis, Roche, Servier. Y. Metaxas: Financial Interests, Institutional, Research Grant: Bristol Myers Squibb. All other authors have declared no conflicts of interest.

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