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Poster Display

7P - Unveiling the prognostic significance of protein expression in advanced high-grade serous ovarian cancer: A comparative study between long-term survivors and early mortal patients

Date

20 Jun 2024

Session

Poster Display

Presenters

Ji-Won Ryu

Citation

Annals of Oncology (2024) 9 (suppl_5): 1-7. 10.1016/esmoop/esmoop103497

Authors

J. Ryu, J. Kim, J. Kim, H. Shin

Author affiliations

  • Gangnam Severance Hospital, Yonsei University College of Medicine, Seoul/KR

Resources

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Abstract 7P

Background

High-grade serous ovarian cancer, despite its high lethality, lacks reliable biomarkers for predicting poor prognosis, and Limited progression has been made in personalized treatment. Genomic profile-based targeted therapy has not met expectations, as genomic alterations alone do not exclusively determine cancer cell phenotypes. Protein expression critically influences cellular processes. Recognizing proteomic alterations is even more crucial. This study proposes a novel technique, utilizing statistical deviation and machine- learning to select protein factors determining ovarian cancer prognosis.

Methods

In advanced high-grade serous ovarian cancer patients, divided into two groups with very good (n=23) and poor prognoses (n=24), proteins were extracted from fresh frozen tissue and subjected to proximity extension assay (PEA). We explored a novel approach called AI-based machine learning to identify key proteins that could distinguish between groups with good and poor prognoses. Proteins were validated by immunohistochemistry (IHC) staining and cell proliferation assay, transwell migration assay, and Boyden chamber invasion assay.

Results

We explored a novel approach called AI-based machine learning to identify key proteins that could distinguish between groups with good and poor prognoses. By developing a model, we found that high levels of NPTN and PPM1A indicated a poor prognosis group, demonstrating remarkably high efficacy (Precision 0.857, Recall 0.818, F1-score 0.893). After IHC of NPTN and PPM1A in a tissue microarray (TMA), survival analysis showed that survival decreased when the expression was high. In vitro experiments with NPTN and PPM1A knockdown showed reduced cell proliferation, migration, and invasion.

Conclusions

Our results suggest that it is feasible to select factors with significant differences between prognostic groups, particularly those that are amenable to clustering based on identified proteins. The research highlights the potential of proteomic markers to guide personalized therapeutic strategies to improve patient outcomes.

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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