Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster Display

64P - Response to PARPi in advanced high-grade serous ovarian cancer (HGSOC) based on the location and type of BRCA mutation: Real-world data from a Spanish tertiary university hospital

Date

20 Jun 2024

Session

Poster Display

Presenters

Isabel Miras Rodriguez

Citation

Annals of Oncology (2024) 9 (suppl_5): 1-19. 10.1016/esmoop/esmoop103501

Authors

I. Miras Rodriguez, P. Estevez Garcia

Author affiliations

  • Hospital Universitario Virgen del Rocio, Seville/ES

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 64P

Background

The presence of a BRCA mutation (BRCAm) is the only biomarker of response to treatment with platinum and PARP inhibitors (PARPi) validated in HGSOC. However, the location of the BRCAm and whether it is germline (gBRCAm) or somatic (sBRCAm) can affect the response to treatment and progression-free survival (PFS). Determining the influence of these factors is necessary to optimize treatment strategies.

Methods

Retrospective observational study of stage III-IV BRCAm or HRD+ HGSOC patients (pts) diagnosed in our institution from 01/2015 to 06/2023. Functional domains (FD) of BRCA1 were defined as RING, DNA-BD or BRCA1 C terminus (BRCT) and FD of BRCA2 were PALB2, RAD51-BD and DNA-BD. Presence of BRCA1/2 mutation was determined using NGS.

Results

Eighty-one pts were included: 32 BRCA1m (21 germline), 29 BRCA2m (12 germline) and 20 BRCAwt/HRD+ (7 germline: 2 PALB2m, 2 RAD51Cm, 2 RAD51Dm, 1 BRIP1m). 42 pts received first-line PARPi (24 olaparib, 8 niraparib and 10 bevacizumab/olaparib). Of the pts who received olaparib, 11 had disease progression/relapse (2 after the end of treatment, 2 after stopping treatment due to toxicity and 7 during it). Pts with a shortest time to progression (3-11 months (m)) had sBRCA1m and/or mutations in the RING or BRCT domains of BRCA1, and they achieved lower overall survival (OS) than expected in BRCAm pts due to poor response to successive treatments. Four pts who received niraparib progressed to treatment, all of them during it (1 BRCAwt/HRD+), and sBRCA1m pts showed worse prognosis. Statistical analysis indicates a positive trend in OS for pts with a missense BRCAm. 12 pts received olaparib in second line of treatment, and 5 of them showed disease progression (2 pts during treatment: 1 with BRCA1 nonsense mutation showed PFS of 3 m). Pts in response reached a median treatment time of 81’5 m. 1 of 3 pts who received niraparib progressed during treatment (showing mutation in the RING domain of BRCA1). Median follow-up was 32’5 m (1-123).

Conclusions

In our study, mutation on BRCA1 RING or BRCT showed a poor response to PARPi and lower survival consistenly with previous reports, highlighting among them somatic mutations.

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

I. Miras Rodriguez: Financial Interests, Personal, Other, Travel grant: MSD, Lilly, Gilead. P. Estevez Garcia: Financial Interests, Personal, Advisory Role: GSK, AstraZeneca, Clovis, MSD; Financial Interests, Institutional, Research Grant: GSK; Financial Interests, Personal, Other, Travel grant: MSD, AstraZeneca, GSK, PharmaMar, Clovis.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.