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Poster Display

17P - Hormone receptor expression outperforms molecular class in predicting endometrial cancer risk pre-operatively

Date

20 Jun 2024

Session

Poster Display

Presenters

Hege Berg

Citation

Annals of Oncology (2024) 9 (suppl_5): 1-7. 10.1016/esmoop/esmoop103497

Authors

H. Berg1, H. Lien1, M. Hjelmeland1, O. Bozickovic1, K. Woie2, I. Haldorsen3, J. Trovik1

Author affiliations

  • 1 University of Bergen, Bergen/NO
  • 2 Haukeland University Hospital, Bergen/NO
  • 3 Helse Bergen - Haukeland University Hospital, Bergen/NO

Resources

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Abstract 17P

Background

Pre-operative histologic subtype and deep myometrial infiltration at magnetic resonance imaging are strong predictors of high-risk disease in endometrial cancer (EC). Whether molecular subtype in combination with hormone receptor status can refine conventional risk stratification is uncertain.

Methods

A prospectively collected EC cohort including 446 patients was molecularly subtyped using surrogate markers and the WHO-endorsed classification algorithm. Median follow-up was 6.4 years. Estrogen- and progesterone receptor (ER and PR) status was investigated by IHC and scored using the staining index method. Uni- and multivariate analyses to predict disease-specific survival (DSS) were performed. The multivariate model included patient age, preoperative risk groups, molecular subtypes and combined ER/PR status.

Results

Patients were classified as POLE (9%), MMR-D (29%), copy-number low (46%) and copy-number high (16%). Loss of ER and/or PR expression was found in 36% of the tumors. Both molecular type and dichotomized ER/PR expression associated with DSS in univariate analyses (p < 0.001). However, after adjusting for preoperative risk group, loss of ER/PR outperforms molecular class for predicting poor DSS (ER/PR: p = 0.004, MolClass: p > 0.05).

Conclusions

Preoperative loss of ER/PR predicts poor prognosis and outperforms molecular class for improving risk stratification of EC patients.

Legal entity responsible for the study

University of Bergen.

Funding

Norwegian Cancer Society, University of Bergen, Helse Vest, The Research Council of Norway.

Disclosure

All authors have declared no conflicts of interest.

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