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Poster Display session

171P - A phase II study of ABSK-011 plus atezolizumab in patients with advanced hepatocellular carcinoma (HCC)

Date

27 Jun 2024

Session

Poster Display session

Presenters

Xiao-Ping Chen

Citation

Annals of Oncology (2024) 35 (suppl_1): S75-S93. 10.1016/annonc/annonc1478

Authors

X. Chen1, Y. Zhang2, L. Zhao3, J. Wu4, T. Liang5, J. Cai6, X. Feng7, Y. Qin8, J. Wang9, C. Hou10, J. Lu11, Z. Yan12, H. Wang12, H. Li12, Q. Cheng13

Author affiliations

  • 1 Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan/CN
  • 2 Harbin Medical University Cancer Hospital, Harbin/CN
  • 3 Shandong Cancer Hospital Affiliated to Shandong University, Jinan/CN
  • 4 The Second Affiliated Hospital of Nanchang University, Nanchang/CN
  • 5 Cancer Hospital of the University of Chinese Academy of Sciences/ Zhejiang Cancer Hospital, Hangzhou/CN
  • 6 Cancer Hospital Chinese Academy of Medical Sciences, Beijing/CN
  • 7 Sichuan Cancer Hospital and Institute/The Affiliated Cancer Hospital School of Medicine, UESTC, Chengdu/CN
  • 8 The First Affiliated Hospital of Zhengzhou University, Zhengzhou/CN
  • 9 Affiliated Hospital of Jining Medical College, Jining/CN
  • 10 Jilin Guowen Hospital, changchun/CN
  • 11 Tangdu Hospital, Fourth Military Medical University, xian/CN
  • 12 Abbisko Therapeutics Co., Ltd., Shanghai/CN
  • 13 Tongji Hospital affiliated to Tongji Medical College of Huazhong, Chibi City/CN

Resources

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Abstract 171P

Background

FGF19 overexpression (+) is in ∼ 30% of HCC. ABSK-011, a highly selective and potent FGFR4i, has demonstrated promising response rate and manageable safety profile for FGF19+ HCC in phase 1 study. This phase 2 open-label study (NCT05441475) is the first study to evaluate the safety and preliminary efficacy of FGFR4i plus immune checkpoint inhibitor (ICI) in FGF19+ HCC.

Methods

ABSK-011 was given QD or BID orally in 21-day cycles and atezolizumab (atezo) was given 1200 mg IV Q3W. Patients (pts) ≥18 years old with advanced HCC, ECOG PS≤1, and adequate organ function were eligible. Tumor assessments were every 6 weeks.

Results

As of 18 Mar 2024, 36 pts were dosed with ABSK-011 180 mg QD (N=6), 320 mg QD (N=6), 160 mg BID (N=8) and 220 mg BID (N=16). Among the 36 pts, the mean age was 51.2 years old with 77.8% male, 63.9% ECOG PS 1, 83.3% with prior therapies. DLT was observed in only 1 pt (160 mg BID): alanine aminotransferase (ALT) and aspartate aminotransferase (AST) increased with blood bilirubin increased and alkaline phosphatase increased. Safety results indicated that 30 pts (83.3%) experienced treatment-related adverse events (TRAEs) for ABSK-011 or atezo, and grade 3 TRAEs reported in ≥2 pts included ALT increased (6/36, 16.7%) and AST increased (3/36, 8.3%), majority of which were transient and improved after dose interruption and/or supportive care. No grade 4/5 TRAEs. Four pts (11.1%) reported serious TRAEs, including ALT increased (3) / AST increased (2) / blood bilirubin increased (2) and haematochezia (1). Seven partial responses were reported by investigator assessment based on RECIST v1.1, including 2 responders (1 FGF19+) in 320 mg QD and 5 (all FGF19+) in 220 mg BID among the 28 efficacy-evaluable pts. 220 mg BID dosing showed encouraging efficacy with an overall response rate of 55.6% (5/9) in FGF19+ HCC. The longest duration of response (DOR) among all dose levels was 15.4 m and median DOR is not yet mature.

Conclusions

ABSK-011 in combination with atezolizumab showed encouraging anti-tumor activity and manageable safety profile which supports further development of the regimen in FGF19+ HCC and exploration of ABSK-011 based double/triple combinations in earlier lines.

Clinical trial identification

NCT05441475.

Legal entity responsible for the study

Abbisko Therapeutics Co., Ltd.

Funding

Abbisko Therapeutics Co., Ltd.

Disclosure

All authors have declared no conflicts of interest.

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