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Poster session 13

483P - Pre-clinical and early clinical assessment of the safety and anti-tumor activity of RP12146, a PARP1/2 inhibitor in solid tumors

Date

10 Sep 2022

Session

Poster session 13

Topics

Pathology/Molecular Biology;  Targeted Therapy

Tumour Site

Breast Cancer;  Ovarian Cancer;  Small Cell Lung Cancer;  Prostate Cancer;  Colon and Rectal Cancer

Presenters

Piotr Tomczak

Citation

Annals of Oncology (2022) 33 (suppl_7): S197-S224. 10.1016/annonc/annonc1049

Authors

P. Tomczak1, M. Kwiatek2, D. Chraniuk3, M. Kmiecik3, I. Lugowska4, M. Chełstowska4, S. Eleswarapu5, S. Veeraraghavan6, K. Routhu7, P. Barde7, A. Nair7

Author affiliations

  • 1 Chemotherapy, Pratia Poznań Medical Center, 60-569 - Poznan/PL
  • 2 Medical Oncology Dept., Pratia Poznań Medical Center, 60512 - Poznan/PL
  • 3 Medical Center, Clinical Trials Site Nasz Lekarz, 87-100 - Toruń/PL
  • 4 Department Of Early Phase Clinical Trials, Maria Sklodowska-Curie National Research Institute of Oncology, 02-781 - Warsaw/PL
  • 5 Biology, Incozen Therapeutics Pvt. Ltd., 500072 - Hyderabad/IN
  • 6 Dmpk, Incozen Therapeutics Pvt. Ltd., 500072 - Hyderabad/IN
  • 7 Clinical Research And Development, Rhizen Pharmaceuticals AG, 4052 - Basel/CH

Resources

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Abstract 483P

Background

RP12146 is an orally bioavailable, potent small-molecule inhibitor of poly (ADP-ribose) polymerase (PARP) 1 and PARP 2 with an IC50 of 0.62nM and 0.44nM respectively and with PARP trapping activity. A phase I/Ib study of RP12146 as a single agent is underway to determine the safety, pharmacokinetics, and anti-tumor activity.

Methods

The phase I/Ib study is designed as a two-part study. Part 1 is a dose-escalation, 3+3 design, MTD determination study and will enroll pts who have tumors that are known to harbour DNA repair deficiencies. Part 2 is dose expansion at the MTD (or optimal dose) and will enroll ovarian, breast cancer, and prostate cancer pts with a confirmed deleterious HRR mutation. Thirty-six pts (12 pts in each expansion group) are planned to be enrolled. Safety is the primary outcome and ORR, CBR, and PFS assessed using RECIST v1.1, would be the secondary outcomes. Biomarker analysis includes the measurement of gammaH2AX in PMBCs.

Results

RP12146 exhibited anti-tumor potential with TGI of 28% as a single agent and 95.19% with gemcitabine combination in the OVCAR-3 xenograft model. In the phase I study, as on 27 Apr 2022, 9 pts of median age 68 years and with 3 median prior therapies have been enrolled at 3 dose levels (100 mg QD, 200 and 400 mg BID) in the dose escalation part. Tumor types included biliary tract, colorectal, breast, prostate, and ovarian cancers. No related TEAEs were reported except for a mild event of neuropathy. None of the patients reported related Grade 3/ 4 AEs. No DLT’s have been reported thus far. None of the pts developed related SAEs or discontinued due to an AE. An ovarian cancer patient who had CDK12 mutation has shown stable disease at C3D1. Five out of 9 patients (55%) are ongoing in the study. RP12146 exhibited rapid absorption achieving maximum concentrations in about 1 hr, with an elimination half-life of ∼ 5 hrs.

Conclusions

RP12146 inhibited tumor growth as a single agent and in combination with gemcitabine in xenograft model of ovarian cancer. In the on-going clinical study, RP12146 has been well-tolerated in the first two cohorts and further evaluation at higher doses in dose escalation is underway. Updated safety and preliminary efficacy data will be provided at the time of presentation.

Clinical trial identification

NCT05002868.

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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