Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster session 13

473P - Phase I study of GFH018, a small molecular TGF-βRI inhibitor, in patients (pts) with advanced solid tumors

Date

10 Sep 2022

Session

Poster session 13

Topics

Clinical Research

Tumour Site

Presenters

Ye Guo

Citation

Annals of Oncology (2022) 33 (suppl_7): S197-S224. 10.1016/annonc/annonc1049

Authors

Y. Guo1, W. Zishu2, H. Zhou2, H. Pan3, W. han3, Y. Deng4, Y. zhang5, H. Ren6, H. Zhang7, S. Wang7, Y. Zhang7, J. Li8

Author affiliations

  • 1 Department Of Medical Oncology, Shanghai East Hospital (South Division), 200120 - Shanghai/CN
  • 2 Department Of Medical Oncology, The First Affiliated Hospital of Bengbu Medical College, 233004 - Bengbu/CN
  • 3 Department Of Medical Oncology, Sir Run Run Run Shaw Hospital, Zhejiang University School of Medicine, 310016 - Hangzhou/CN
  • 4 Department Of Medical Oncology, The Sixth Affiliated Hospital, Sun Yat-sen University, 510655 - Guangzhou/CN
  • 5 Gastrointestinal Medical Oncology, Harbin Medical University Cancer Hospital, 150040 - Harbin/CN
  • 6 Department Of Biology, GenFleet Therapeutics (Shanghai) Inc., Shanghai/CN
  • 7 Clinical Department, GenFleet Therapeutics (Shanghai) Inc., Shanghai/CN
  • 8 Department Of Medical Oncology, Shanghai East Hospital (South Division), Shanghai/CN

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 473P

Background

TGF-β signaling pathway activation promotes tumor metastasis and progression, mediates epithelial-mesenchymal transmission (EMT) suppressing immunosurveillance in advanced tumors. GFH018, a novel small molecule that inhibits TGF-βRI kinase, blocks TGF-β signaling transduction thus inhibits tumor progression. In preclinical models, GFH018 showed significant anti-tumor activity as monotherapy and in combination with an anti-PD-1 antibody. This phase I study will assess the safety, pharmacokinetics (PK), and preliminary efficacy of GFH018 monotherapy in pts with advanced solid tumors.

Methods

This is an open-label, multicenter study comprising of a modified 3 + 3 dose escalation part followed by an expansion part. The starting dose was 5 mg and up to 8 cohorts were planned. Eligible pts with advanced solid tumors failed to standard therapies were administrated with GFH018 BID, 14d on/14d off in 28-day cycles. AEs were graded per NCI-CTCAE v5.0. PK analysis was performed using a non-compartmental method. Efficacy was evaluated per RECIST 1.1.

Results

As of Jan 25, 2022, 39 pts (5 mg [n=4]; 10 mg [n=3]; 20mg [n=4]; 30 mg [n=7]; 40, 50 mg [n=4 each]; 65 mg [n=6] and 85 mg [n=7]) were sequentially enrolled in the dose escalation part. The median lines of prior therapy were ≥ 3. No DLT was observed and the MTD was not reached. No pts discontinued due to AE. 35 pts (89.7%) had at least one related AE and 2 (5.1%) experienced ≥ grade (G) 3 related AE. The most common related AEs (all G/≥G3) were proteinuria (28.2%/2.6%), AST increased (15.4%/0), ALT increased (12.8%/0), anemia (12.8%/0), lymphocyte count decreased (10.3%/2.6%), ALP increased (10.3%/0), and LDH increased (10.3%/0). PK of GFH018 was linear and dose-independent with mean half-life in the range of 3.11 h- 8.30 h. Accumulation in PK exposure was limited post multiple dosing. Of 24 evaluable pts, 5 achieved stable disease (SD). A pt with thymic carcinoma receiving 50 mg achieved a durable SD with tumor shrinkage (maximum lesion decreased by 18.4%) and has stayed on treatment for 185 days as of the data cut-off date.

Conclusions

GFH018 shows a favorable safety profile and is now being tested in combination with an anti-PD-1 antibody in patients with advanced malignancies. Further studies are warranted.

Clinical trial identification

NCT05051241.

Editorial acknowledgement

Legal entity responsible for the study

GenFleet Therapeutics (Zhejiang) Co., Ltd.

Funding

GenFleet Therapeutics (Zhejiang) Co., Ltd.

Disclosure

H. Ren, H. Zhang, S. Wang, Y. Zhang: Financial Interests, Personal and Institutional, Full or part-time Employment: GenFleet Therapeutics, Inc. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.