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Poster session 08

378P - Liquid biopsy detects early molecular response and predicts benefit to first-line chemotherapy plus cetuximab in metastatic colorectal cancer: PLATFORM-B study

Date

10 Sep 2022

Session

Poster session 08

Topics

Tumour Site

Colon and Rectal Cancer

Presenters

JOANA Vidal Barrull

Citation

Annals of Oncology (2022) 33 (suppl_7): S136-S196. 10.1016/annonc/annonc1048

Authors

J. Vidal Barrull1, M. Nieva Munoz1, C. Fernandez-Rodriguez2, P. García-Alfonso3, D. Paez4, V. Alonso-Orduna5, M.T. Cano Osuna6, C. Santos Vivas7, G. Duran8, M.E. Elez Fernandez9, J.L. Manzano Mozo10, R. Garcia-Carbonero11, E. Pineda12, J. Sastre Varela13, F. Rivera Herrero14, B. Bellosillo Paricio2, J. Tabernero9, E. Aranda Aguilar6, R. Salazar7, C. Montagut Viladot1

Author affiliations

  • 1 Medical Oncology Dept., Hospital del Mar, Institut Mar Investigacions Mèdiques (IMIM), Universitat Pompeu Fabra, CIBERONC, 8003 - Barcelona/ES
  • 2 Pathology Department, Hospital del Mar - IMIM, 8003 - Barcelona/ES
  • 3 Medical Oncology Department, Hospital General Universitario Gregorio Marañon, 28007 - Madrid/ES
  • 4 Dept. Medical Oncology, Hospital de la Santa Creu i Sant Pau, U705. CIBERER, 08025 - Barcelona/ES
  • 5 Medical Oncology Service, Hospital Universitario Miguel Servet, 50009 - Zaragoza/ES
  • 6 Medical Oncology, IMIBIC, Reina Sofía Hospital, University of Cordoba, CIBERONC, 14004 - Cordoba/ES
  • 7 Medical Oncology Department, Catalan Institute of Oncology (ICO), Bellvitge Biomedical Research Institute (IDIBELL), CIBERONC, 08908 - L'Hospitalet de Llobregat/ES
  • 8 Medical Oncology, Unidad de Gestión Clínica Intercentros de Oncología Médica. Hospitales Universitarios Regional y Virgen de la Victoria, IBIMA, 29010 - Malaga/ES
  • 9 Medical Oncology Dept., Vall d’Hebron Barcelona Hospital Campus, Vall d’Hebron Institute of Oncology (VHIO), Universitat Autònoma de Barcelona, CIBERONC, 8035 - Barcelona/ES
  • 10 Medical Oncology Department, ICO - Institut Català d'Oncologia Badalona (Hospital Universitario Germans Trias i Pujol), 08916 - Badalona/ES
  • 11 Medical Oncology Department, Hospital Universitario 12 de Octubre, Instituto de Investigación Sanitaria Hospital 12 de Octubre (Imas12), UCM, 28041 - Madrid/ES
  • 12 Medical Oncology Department, Hospital Clinic y Provincial de Barcelona, 08036 - Barcelona/ES
  • 13 Medical Oncology, Hospital Universitario Clínico San Carlos, CIBERONC, 28040 - Madrid/ES
  • 14 Medical Oncology, Hospital Marqués de Valdecilla, IDIVAL, 39008 - Santander/ES

Resources

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Abstract 378P

Background

Chemotherapy plus anti-EGFR therapy is the standard first-line treatment in RAS wild-type (wt) metastatic colorectal cancer (mCRC). The utility of ctDNA sequencing (liquid biopsy) to assess early molecular response to chemotherapy plus anti-EGFR therapy before radiological progression is yet to be determined, and should integrate information on changes in total tumor burden and early emergence of resistant sub-clones.

Methods

We conducted a prospective multicentric observational study of patients with tissue RAS wt mCRC treated with first-line chemotherapy plus cetuximab undergoing sequential liquid biopsies. Baseline and early (C3) ctDNA were analyzed by a comprehensive NGS-based approach. Trunk mutations were assessed as a surrogate marker of total tumor burden and RAS/BRAF/MEK/EGFR-ECD/MAP2K1 were considered mutations of resistance.

Results

One hundred patients were included. Decrease in ctDNA trunk mutations between baseline and C3 correlated with progression-free survival (PFS) (HR= 0.23 P=0.001). RAS/BRAF were the only mutations of resistance detected at C3, while other coexisting and sub-clonal mutations of acquired resistance -mainly mutations in EGFR-ECD- emerged later during anti-EGFR therapy. An increase in the relative fraction of RAS/BRAF at C3 was associated with shorter PFS (HR= 10.5, P< 0.001), and predicted clonal expansion of the specific RAS/BRAF mutation in sequential liquid biopsies. Combined analysis of trunk and resistant mutations at C3 showed that patients with “early molecular response” (decrease in trunk and decrease in mutations of resistance at C3) had better response (77.5% vs. 25%, P=0.008) and longer PFS (HR=0.18, P< 0.001) compared to patients with “early molecular progression” (increase in trunk and/or increase in mutations of resistance).

Conclusions

Liquid biopsy detects early molecular response and predicts benefit to chemotherapy plus cetuximab. A comprehensive NGS-based approach is necessary to integrate information on total disease burden and resistance mutations. This sets the rationale for clinical trials to evaluate early ctDNA-guided treatment decisions in first-line mCRC.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

Spanish Cooperative Group for the Treatment of Digestive Tumors (TTD).

Funding

Instituto de Salud Carlos III – FEDER Grant PI18/00031 and Fundacion CRIS Excellence 19-30 Grant.

Disclosure

V. Alonso-Orduna: Financial Interests, Personal, Speaker’s Bureau: Hoffman La-Roche, Merck-Serono, Sanofi, Amgen; Financial Interests, Personal, Invited Speaker: Servier; Financial Interests, Personal, Advisory Board: Amgen, Merck-Serono; Financial Interests, Personal, Other, Support for attending meetings and/or travel: Merck-Serono, Hoffman La-Roche. C. Santos Vivas: Financial Interests, Personal, Speaker’s Bureau, lectures, presentations, speakers bureaus, manuscript writing or educational events: Pierre-Fabre; Financial Interests, Personal, Expert Testimony: Sanofi, Amgen ; Financial Interests, Personal, Other, attending meetings and/or travel: Amgen; Financial Interests, Personal, Invited Speaker, attending meetings and/or travel: Merck. M.E. Elez Fernandez: Financial Interests, Institutional, Research Grant: AbbVie, Amgen, Array Pharmaceuticals, AstraZeneca, Boehringer Ingelheim, Bristol-Myers Squibb, GlaxoSmithKline, Hoffman La-Roche, Medimmune, Merck Serono, MSD, Novartis, Pierre-Fabre, Sanofi Aventis; Financial Interests, Personal, Advisory Board: Amgen, Array Biopharma, Bayer, Bristol Myers Squibb, Hoffman La - Roche, Merck Serono, Sanofi, Servier; Financial Interests, Personal, Other, Travel, Accommodations, Expenses: Amgen, Servier, Array Biopharma, Merck Serono, Sanofi, Bristol Myers Squibb, Roche. R. Garcia-Carbonero: Financial Interests, Personal, Funding, for continuous medical education: AAA, Advanz Pharma, Amgen, Bayer, BMS, Hutchmed, Ipsen, Merck, Midatech Pharma, MSD, Novartis, PharmaMar, Pierre Fabre, Roche, Servier , Sanofi; Financial Interests, Personal, Other, research support: Pfizer, BMS , MSD. F. Rivera Herrero: Financial Interests, Personal, Research Grant: Sanofi, Roche, MSD, BMS, Bayer, Lilly, Astellas, Servier, Merck-Serono, Amgen, Celgene; Financial Interests, Personal, Speaker’s Bureau: Sanofi, Roche, MSD, BMS, Bayer, Lilly, Astellas, Servier, Merck-Serono, Amgen, Celgene; Financial Interests, Personal, Other, attending meetings and/or travel: Sanofi, Roche, MSD, BMS, Bayer, Lilly, Astellas, Servier, Merck-Serono, Amgen, Celgene ; Financial Interests, Personal, Advisory Board: Sanofi, Roche, MSD, BMS, Bayer, Lilly, Astellas, Servier, Merck-Serono, Amgen, Celgene. E. Aranda Aguilar: Financial Interests, Personal, Advisory Role: Amgen, Bayer, Bristol Myers Squibb, Merck, Roche, Sanofi. All other authors have declared no conflicts of interest.

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