Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster session 13

756P - Glypican-3 (GPC3) and NKp46 directed FLEX-NK engager antibody (CYT-303) recruits natural killer (NK) cells to tumors in a preclinical hepatocellular carcinoma (HCC) mouse model

Date

10 Sep 2022

Session

Poster session 13

Topics

Cancer Biology;  Tumour Immunology;  Molecular Oncology;  Immunotherapy

Tumour Site

Hepatobiliary Cancers

Presenters

Antonio Arulanandam

Citation

Annals of Oncology (2022) 33 (suppl_7): S331-S355. 10.1016/annonc/annonc1058

Authors

A. Arulanandam1, H. Potu1, V. Khairnar1, D. Zou1, M. Triggiano1, N. Dilmac1, L. Lin1, H. Chang1, A. Welch1, O. Mandelboim2, Y. Ilan3, D. Teper1, S. Frankel1, J. Kadouche1, W. Li1

Author affiliations

  • 1 Research, Cytovia Therapeutics, 33180 - Aventura/US
  • 2 Immunology And Cancer Research, Hebrew University of Jerusalem, 9112102 - Jersualem/IL
  • 3 Gastroenterology, Hadassah-Hebrew University Medical Center, 91120 - Jerusalem/IL

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 756P

Background

GPC3 is an oncofetal antigen that is highly expressed in HCC while minimally expressed in adult normal tissues except placenta. CYT-303 is a multifunctional bispecific NK engager targeting NK cell activating receptor NKp46 and GPC3 expressed on tumor cells constructed with Cytovia’s Flex-NK scaffold with a fully functional Fc. Previously, we reported CYT-303 in vitro treatment of Hep3B tumor cells demonstrated cytolysis and cytokine production as well as in vivo tumor growth inhibition when combined with both PBNK and iNK cells. Here we further characterized the CYT-303 dose-response for tumor growth inhibition in vivo and its influence on NK cell distribution.

Methods

CYT -303 pharmacokinetics was evaluated in tumor-bearing mice. Anti-tumor efficacy and dose response of CYT-303 in the presence of human PBNKs or iNKs was evaluated in NSG-IL15 mice bearing subcutaneous Hep3B tumors. Biodistribution of the NK cells were analyzed by flow cytometry.

Results

CYT-303 pharmacokinetics in tumor bearing mice was similar to normal mice suggesting no target mediated clearance. In the Hep3B tumor model in PBNK or iNK injected NSG-hIL-15 mice, CYT-303 showed dose dependent tumor growth inhibition compared to control hIgG1 treated mice. Consistent with CYT-303 tumor growth inhibition, increased NK cells were detected in the tumor compared to control animals. Blood NK cells in CYT-303 treated animals were significantly lower compared to IgG1 isotype control treated mice suggesting CYT-303 could facilitate NK cell trafficking from blood into the tumor. Blood AFP (alpha fetoprotein), a biomarker in HCC, decreased with CYT-303 tumor growth inhibition.

Conclusions

Pharmacologically active CYT-303 doses were identified. CYT-303 can help recruit NK cells to the tumor site to achieve tumor growth inhibition.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

Cytovia Therapeutics.

Funding

Cytovia Therapeutics.

Disclosure

A. Arulanandam: Financial Interests, Personal, Stocks/Shares: Cytovia Therapeutics. A. Welch: Financial Interests, Personal, Stocks/Shares: Cytovia Therapeutics. D. Teper: Financial Interests, Personal, Stocks/Shares: Cytovia Therapeutics. S. Frankel: Financial Interests, Personal, Stocks/Shares: Cytovia Therapeutics. J. Kadouche: Financial Interests, Personal, Stocks/Shares: Cytovia Therapeutics. W. Li: Financial Interests, Personal, Stocks/Shares: Cytovia Therapeutics. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.