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Poster session 16

1177P - Clinical implications of TSC1/TSC2 variants on immunotherapy in NSCLC patients

Date

10 Sep 2022

Session

Poster session 16

Topics

Tumour Site

Non-Small Cell Lung Cancer

Presenters

Lijuan Chen

Citation

Annals of Oncology (2022) 33 (suppl_7): S448-S554. 10.1016/annonc/annonc1064

Authors

L. Chen1, S. Yuan1, H. Chen1, X. Shi2, D. Chen3, J. Hu4, Z. Yang5, S. Zhang1, C. Chen6, L. Mei3

Author affiliations

  • 1 Bioinformatics, Shanghai OrigiMed Co., Ltd, 201114 - Shanghai/CN
  • 2 Department Of Molecular Pathology, Shanghai OrigiMed Co., Ltd, 201114 - Shanghai/CN
  • 3 Pathology, Shanghai OrigiMed Co., Ltd, 201114 - Shanghai/CN
  • 4 Clinical Interpretation Department, Shanghai OrigiMed Co., Ltd, 201114 - Shanghai/CN
  • 5 Clinical Database Department, Shanghai OrigiMed Co., Ltd, 201114 - Shanghai/CN
  • 6 Sequencing, Shanghai OrigiMed Co., Ltd, 201114 - Shanghai/CN

Resources

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Abstract 1177P

Background

TSC1 and TSC2 are important tumor suppressor genes, but their research in NSCLC are still limited, especially their association with immunotherapy. This study aims to investigate the relationship of TSC1/TSC2 variants and immunotherapy in Chinese NSCLC patients.

Methods

Retrospective patients were collected, short variants, copy number variations, gene rearrangements and Tumor Mutation Burden (TMB) were analyzed following CAP/CLIA-certificated workflows. Wilcoxon Rank Sum Test were applied to significance statistics.

Results

TSC1/2 variants were detected in 3.3% patients (n=10,182), with 150 (44.4%) in TSC1 and 192 (56.8%) in TSC2. In TSC1/2-altered patients, 58.5% were male, 47.6% were at advanced stage (stage III or IV) and the median age was 61 years old (range, 29-81 years old). In TSC1 altered cases, the median TMB was 4.7 muts/Mb, ranging from 0 to 88.3 muts/Mb. In TSC2 altered cases, the median TMB was 7.7 muts/Mb, ranging from 0 to 128.7 muts/Mb. Interestingly, TMB in TSC1/TSC2 altered patients and other patients showed significant difference, with a p-value of 1.9x10-09 (11.29 vs 7.35 muts/Mb in mean), with 125 (37.0%) showed TMB-H. Moreover, TSC2 and TSC1 patients showed significant difference in TMB (p-value=0.0007). In TSC1 or TSC2 altered cases, TP53 (53.8%), EGFR (40.8%), BRAF (18.6%), KRAS (14.5%) and CDKN2A (13.0%) were the top 5 co-occurred mutated genes. Furthermore, 248 patients harboring TSC1/TSC2 variants were analyzed by IHC with PD-L1 antibody, 30.6% showed positive (TPS>1%) and recommended immunotherapy treatment. What’s more, we found that in TSC1 cases the advanced stage group showed significant higher PD-L1 positive than early stage group (43.4% vs 17.8%, p-value=9.06e-05), other stages showed no significance.

Conclusions

TSC1/2 variants group showed higher TMB and PD-L1 expression compared to non-TSC1/2 variants group. This information may suggest the potential utilities of TSC1/2 mutation status on immunotherapy in NSCLC.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Shanghai OrigiMed Co., Ltd.

Disclosure

All authors have declared no conflicts of interest.

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