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Poster session 10

922P - Automated and rapid KRAS mutation testing in non-small cell lung cancer

Date

10 Sep 2022

Session

Poster session 10

Topics

Laboratory Diagnostics;  Pathology/Molecular Biology;  Molecular Oncology;  Targeted Therapy

Tumour Site

Non-Small Cell Lung Cancer

Presenters

Gonzalo Recondo

Citation

Annals of Oncology (2022) 33 (suppl_7): S417-S426. 10.1016/annonc/annonc1061

Authors

G. Recondo1, S. Akselrad2, F. Jauk3, P. Teves2, J. Pandolfi3, L. Basbus4, C.A. Franco Cortes3, H. Garcia Rivello3, R. Sanchez Marull2, Y. Powazniak5, J. Calderazzo2, M.V. Bluthgen6, S.N. Sena7, A. Perfetti8, P.P. Levit8, N. Castagneris9, M.M. Rizzo10, L. Lupinacci11, J.N. Minatta12

Author affiliations

  • 1 Medical Oncology, Centro de Educacion Medica e Investigaciones Clinicas (CEMIC), C1431FWO - Buenos Aires/AR
  • 2 Pathology, CEMIC - Centro de Educacion Medica e Investigacones Clinicas Dr Norberto Quirno, C1431FWO - Buenos Aires/AR
  • 3 Pathology, Hospital Italiano de Buenos Aires, C1199ABB - Buenos Aires/AR
  • 4 Oncology, Hospital Italiano de Buenos Aires, C1414 - Buenos Aires/AR
  • 5 Precision Medicine Institute, Centro de Educacion Medica e Investigaciones Clinicas (CEMIC), 1423 - Buenos Aires/AR
  • 6 Clinical Oncology Department, Hospital Aleman, C1118AAT - Buenos Aires/AR
  • 7 Oncologia, Hospital Aleman, C1118AAT - Buenos Aires/AR
  • 8 Caba, Centro Medico Accor, Buenos Aires/AR
  • 9 Oncology Department, Clinica Universitaria Reina Fabiola, X5004FHP - Cordoba/AR
  • 10 Medical Oncology Department, Hospital Universitario Austral, B1629AHJ - Pilar/AR
  • 11 Clinical Oncology Department, Hospital Italiano de Buenos Aires, C1199ABB - Buenos Aires/AR
  • 12 Oncology Department, Hospital Italiano de Buenos Aires, C1414 - Buenos Aires/AR

Resources

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Abstract 922P

Background

Detecting KRAS mutations in tumor samples can be challenging in low-middle-income countries due to limited access to molecular biology laboratories. The Idylla TM KRAS mutation test (IKMT) is an IVD, automated, and rapid qPCR assay to detect KRAS mutations, validated in colorectal cancer. We studied the concordance of the IKMT in KRAS mutation detection, compared with NGS, and performed a prospective KRAS mutation test feasibility study in samples from patients with NSCLC.

Methods

To study the concordance of IKMT to NGS we included lung cancer samples sequenced using the Oncomine Focus Assay (OFA) NGS panel. The positive percentage agreement (PPA) and negative percentage agreements (NPA) were estimated. To study the feasibility of KRAS testing using IKMT we prospectively included patients with stage IV lung cancer with wild-type EGFR, BRAF, ALK, and ROS1 tumors. The Idylla KRAS mutation test was performed according to manufacturer instructions in an Idylla TM platform (Biocartis) in 2 hours.

Results

To study the concordance between IKMT and NGS, 26 KRAS mut and 20 KRAS wt samples were included. The concordance rate was 100% and PPA was 100% in 25 KRAS mutant samples: G12C (N =12), G12V (N = 7), Q61H (N = 3), G12A (N = 1), G12D (N = 1) and G12S (N= 1). The NPA was 100%. In the feasibility study, 135 patients were included, IKMT was successfully done in 131 (97%) samples, two did not meet assay quality control, and two runs were aborted by the system due to cartridge issues. The median tumor cell percentage in the sample was 65% (range: 10-95), median tumor area in tissue slides was 23 mm2 (range:5-500). Among the 131 samples tested, 54 (41%) had a detectable KRAS mutations, including G12C (N = 23, 17.6%), G12V (N =15, 11.5%), G12A (N = 5, 3,8%), G12D (N = 3, 2.3%), G13D (N = 3, 2.3%), G12S (N = 2, 1.5%), Q61H (N =2, 1.5%) and A146P/T/V (N = 1, 0.8%). The rate of KRAS mutation detection was similar across sample size (39.3% in < 25mm2 vs.38.2% in >25mm2, p = 0.9) and biopsy procedure (surgical 45.6% vs.core/FNA 30.9%, p = 0.13).

Conclusions

The Idylla TM KRAS mutation test shows high concordance and agreement with NGS testing to detect KRAS mutations in lung cancer samples. KRAS G12C mutations were found in about 18% of tumors without EGFR, BRAF, ALK, and ROS1 alterations.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Amgen.

Disclosure

G. Recondo: Financial Interests, Advisory Board: Amgen, Bayer, Biocartis, BMS, Merck Serono, MSD, Pfizer, Roche, Takeda; Financial Interests, Research Grant: Amgen, Janssen. M.V. Bluthgen: Financial Interests, Invited Speaker: Roche, MSD, BMS, AstraZeneca, Pfizer, Takeda. A. Perfetti: Financial Interests, Advisory Board: Roche, Pfizer, Boehringer, BMS, MSD, Amgen, Novartis, Boehringer Ingelheim. N. Castagneris: Financial Interests, Invited Speaker: Takeda, BMS, Amgen, Roche, Pfizer, AstraZeneca. L. Lupinacci: Financial Interests, Advisory Board: BMS, Amgen, Roche, Pfizer. J.N. Minatta: Financial Interests, Research Grant: Pfizer; Financial Interests, Advisory Board: Pfizer, MSD, Takeda, Roche, Raffo, Merck, Amgen. All other authors have declared no conflicts of interest.

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