Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster session 13

495TiP - A phase II study evaluating the safety and efficacy of ENV-101 (taladegib) in patients with advanced solid tumors harboring PTCH1 loss of function mutations

Date

10 Sep 2022

Session

Poster session 13

Topics

Clinical Research;  Targeted Therapy;  Molecular Oncology

Tumour Site

Presenters

Jason Luke

Citation

Annals of Oncology (2022) 33 (suppl_7): S197-S224. 10.1016/annonc/annonc1049

Authors

J.J. Luke1, S. Pendyala2, A. Di Francesco3, S. Kim3, L. Dellamary4, S. KC4, H. Humphreys3, M. De Los Rios3, J. Pitman3, J. Hood3, D.S. Hong5

Author affiliations

  • 1 Medical Oncology, University of Pittsburgh Cancer Institute, 15232 - Pittsburgh/US
  • 2 Oncology, Endeavor Biomedicines, 92130 - San Diego/US
  • 3 Clinical Research, Endeavor Biomedicines, 92130 - San Diego/US
  • 4 Other, Endeavor Biomedicines, 92130 - San Diego/US
  • 5 Investigational Cancer Therapeutics Department, The University of Texas MD Anderson Cancer Center, 77035 - Houston/US

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 495TiP

Background

ENV-101 is a Hh pathway inhibitor (Smo inhibitor) originally developed as a treatment for patients with advanced solid tumors. Clinical responses have been reported in patients with gene mutations affecting PTCH1 treated with other Hh pathway inhibitors. Endeavor is studying ENV-101 in advanced cancer patients with solid tumors harboring PTCH1 loss of function mutations. This study aims to evaluate dose as well as the efficacy and safety of ENV-101, a potent Hh pathway inhibitor, in patients with refractory advanced solid tumors characterized by loss of function mutations in the PTCH1 gene. Two distinct mechanisms are responsible for inappropriate and uncontrolled Hh pathway activation in human malignancies: ligand-dependent, due to overexpression of Hh ligand, and ligand-independent, resulting from genetic mutations in pathway components such as Ptch and Smo. The ligand-dependent mediated Hh pathway activation is implicated in a number of malignancies, including basal cell carcinoma (BCC), medulloblastoma, rhabdomyosarcoma, breast cancer, esophageal cancer, gastric cancer, pancreatic cancer, prostate cancer, small cell lung cancer, bladder cancer, oral cancer, and melanoma. Hedgehog pathway activation due to the ligand-independent mechanism involving Ptch1 loss of heterozygosity (LOH) or Smo mutations are primarily reported in medulloblastoma and BCC. However, data indicate that these mutations may be present in other solid tumors as well. In addition, as the Hh pathway is known to be primarily active in embryonic development and is not required for survival in adults, this type of targeted therapy should provide improved outcomes to defined cancer patient populations.

Trial design

This is a Simon 2 stage design that aims to evaluate the efficacy and safety of ENV-101, a potent Hedgehog (Hh) pathway inhibitor, in patients with refractory advanced solid tumors characterized by loss of function (LOF) mutations in the PTCH1 gene. Stage 1 (phase IIa) of this protocol will enroll a total of 44 patients randomized between two dose levels. In the presence of acceptable efficacy, stage 2 (phase IIb) of this protocol will expand enrollment using a single dose level.

Clinical trial identification

NCT05199584 January 20, 2022.

Editorial acknowledgement

Legal entity responsible for the study

Endeavor Biomedicines.

Funding

Endeavor Biomedicines.

Disclosure

All authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.