Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

ePoster Display

1203P - The analysis of ALK fusion variants in 4991 EGFR/MET mutation-negative non-squamous non-small cell lung carcinomas (NSCLCs)

Date

16 Sep 2021

Session

ePoster Display

Topics

Cytotoxic Therapy;  Targeted Therapy;  Cancer Biology;  Translational Research;  Pathology/Molecular Biology

Tumour Site

Non-Small Cell Lung Cancer

Presenters

Evgeny Imyanitov

Citation

Annals of Oncology (2021) 32 (suppl_5): S949-S1039. 10.1016/annonc/annonc729

Authors

E.N. Imyanitov, E. Preobrazhenskaya, A. Romanko, A. Martianov, N. Mitiushkina, V. Tiurin

Author affiliations

  • Department Of Tumor Growth Biology, N.N. Petrov Institute of Oncology, 197758 - St.-Petersburg/RU

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 1203P

Background

IHC- and FISH-based methods of ALK testing do not identify variants of ALK fusions. NGS analysis is expensive, hence its use is still limited. ALK translocations result in increased transcription of the kinase portion of the gene, therefore PCR analysis for unbalanced 5’/3’-end expression is a cost-efficient tool for a comprehensive detection of all variants of ALK rearrangements.

Methods

This study included 4991 EGFR/MET mutation-negative NSCLCs, which were referred for diagnostic purposes to the N.N. Petrov Institute of Oncology (St-Petersburg) between April, 2018 and October, 2020.

Results

744/4991 (14.9%) NSCLCs showed evidences for ALK unbalanced 5’/3’-end expression, although only 427 of them demonstrated a disbalance between the border of the rearrangement (exon 19) and the 3’-portion of the gene. Variant-specific PCR assay, which was designed to detect 20 the most common ALK fusions, revealed translocation in 291/4991 (5.9%) NSCLCs; 266/291 (91.4%) and 278/291 (95.5%) of these ALK-rearranged tumors demonstrated 5’/3’ and ex19/3’unbalanced expression, respectively. We further considered 47 tumors, which appeared the most promising by the test for unbalanced expression and did not carry KRAS mutation. NGS revealed 16 instances of ALK translocations (4 novel (UBCex1-ALKex18, EML4ex13-ALKex3-ALKex20, EML4ex6-ins18bp-ALKex20 and EML4ex5del10-del44ALKex20) variants; 10 tumors with known rare ALK translocations; 2 common variants present in a low fraction of tumor cells). In addition, we subjected to QIAseq RNAscan NGS analysis 32 young-onset NSCLCs, which were negative by PCR ALK/ROS1/RET translocation assays; no NSCLCs with ALK translocations were revealed, although a novel ACTB-ROS1 fusion was observed in a single case.

Conclusions

This study provides a framework for non-expensive and efficient detection of ALK fusions.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Russian Science Foundation (grant 17-75-30027).

Disclosure

All authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.