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ePoster Display

349P - The amount of cancer stem cells and the sensitivity to anticancer drugs in glioblastoma primary cell culture

Date

16 Sep 2021

Session

ePoster Display

Topics

Cancer Biology;  Pathology/Molecular Biology

Tumour Site

Central Nervous System Malignancies;  Head and Neck Cancers

Presenters

Olga Susova

Citation

Annals of Oncology (2021) 32 (suppl_5): S516-S529. 10.1016/annonc/annonc674

Authors

O. Susova1, A. Poletaeva2, A. Lupatov3, I. Kholodenko3, A. Karamysheva1, A.A. Mitrofanov4, D. Naskhletashvili5, T. Nasedkina6, A. Bekyashev4

Author affiliations

  • 1 Department Of Tumor Cell Genetics, N.N. Blokhin NMRCO, 119991 - Moscow/RU
  • 2 Department Of Tumor Cell Genetics, Federal State Budgetary Institution «N.N. Blokhin National Medical Research Center of Oncology» оf the Ministry of Health of the Russian Federation, Moscow/RU
  • 3 Biology Of Cancer Stem Cells, Institute of Biomedical Chemistry, Moscow, 119991 - Moscow/RU
  • 4 N. N. Trapeznikov Research Institute Of Clinical Oncology, Federal State Budgetary Institution «N.N. Blokhin National Medical Research Center of Oncology» оf the Ministry of Health of the Russian Federation, Moscow/RU
  • 5 N. N. Trapeznikov Research Institute Of Clinical Oncology, N.N. Blokhin NMRCO, 119991 - Moscow/RU
  • 6 Laboratory Of Biological Microchips, EIMB-Engelhardt Institute of Molecular Biology-Russian Academy of Sciences, 119991 - Moscow/RU

Resources

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Abstract 349P

Background

It is assumed that aggressiveness and therapy resistance of glioblastoma multiforme (GBM) is determined by its high heterogeneity and presence of cancer stem cells (CSC). The primary glioblastoma cell lines (pGBM lines) could better than the cultured cell lines reflect the tumor heterogeneity and, thus, provide the additional opportunities for personalized medicine. In our study, we compared the response of the pGBM lines and cell glioblastoma lines to chemotherapy drugs with the CSC content in cell cultures.

Methods

Four pGBM lines obtained from patients and standart glioblastoma line U251 were used. The amount of CSCs in cell culture was evaluated with flow cytometry by expression of CD133/CD44 and CD90/CD95. Further, the cultures were characterized by immunostaining with Nestin and Oct4.Chemotherapeutic agents included both drugs, commonly used in clinical practice for glioblastoma treatment and drugs used for therapy of other types of cancer. Cytotoxicity was tested via MTT assay. Proliferation rate was measured through cell counting within 3 following days.

Results

The correlations between CD133/ CD44 and CD90/CD95 expression, proliferation rate, and drug resistance of cell cultures were evaluated. The U251 cell line demonstrated significantly increased sensitivity towards cytostatic agents such as gemcitabinum, cisplatin, irinotecan, doxorubicin as compared to pGBM-lines, although its proliferation rate was not the highest. For all studied GBM-lines, the amount of CD90+/CD95+ cells negatively correlated with rozustin sensitivity. The Gbl24n line was the only cell culture with CD133+ cells (about 10%) and it was 20-25% less sensitive to lomustine. Carfilzomib sensitivity positively correlates with the amount of CD133-/CD44- cells, whereas irinotecan sensitivity has a negative correlation with this parameter.

Conclusions

In total, the pGBM lines are less sensitive to anti-cancer drugs, than the U251 cell line. That might result from heterogeneity of primary culture and the presence of CSC in primary tumor cultures. For several drugs (lomustine), the correlation between the amount of CSC in cell culture and drug sensitivity was found.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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