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ePoster Display

901P - Relationship between p16 and tumor-infiltrating lymphocytes in Peruvian patients with head and neck cancer

Date

16 Sep 2021

Session

ePoster Display

Topics

Tumour Site

Head and Neck Cancers

Presenters

Carlos Castaneda

Citation

Annals of Oncology (2021) 32 (suppl_5): S786-S817. 10.1016/annonc/annonc704

Authors

C.A. Castaneda1, M. Castillo Garcia2, L.A. Bernabe2, J. Sanchez2, K. Tello2, R. Alatrsita2, L. Villa3, L. Sichero3, J. Enciso4, H. Guerra5

Author affiliations

  • 1 Medical Oncology Department, Instituto Nacional de Enfermedades Neoplasicas, INEN, Lima 34 - Lima/PE
  • 2 Research Department, Instituto Nacional de Enfermedades Neoplasicas, INEN, Lima 34 - Lima/PE
  • 3 Radiology And Oncology, Faculdade De Medicina, Instituto do Câncer do Estado de São Paulo, 01000 - Sao Paulo/BR
  • 4 Laboratorio De Cultivo Celular E Inmunología, Universidad Cientifica del Sur, 15067 - Lima/PE
  • 5 Pathology Department, Instituto Nacional de Enfermedades Neoplasicas, INEN, Lima 34 - Lima/PE

Resources

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Abstract 901P

Background

Human papillomavirus-16 (HPV-16) infection in Head and Neck Squamous Cell Carcinoma (HNSCC) has a different prevalence through worldwide regions, it influences prognosis and treatment response and p16 staining is its surrogate biomarker. The density and composition of tumor-infiltrating lymphocytes (TIL) reflect biology and prognosis in different malignancies. We characterize TIL in Peruvian patients with HNSCC. We evaluate the influence of clinicopathological features including p16 & ki67 staining in tumor cells over TIL level.

Methods

CD3, CD8, p16, and Ki67 immunohistochemistry were performed on tissue microarrays (TMA), density of CD3/CD8 positive cells were analyzed with Visiopharm Tissuealign Software, and p16 status and ki67 level were evaluated by a pathologist. TIL was evaluated following the International TIL working group. Statistical analysis was performed with SPSS software.

Results

We included 178 HNSCC who came to the institute from 2005 to 2015 and who have enough archived tumor tissue to prepare TMAs. The median age was 65 y, most were female (56.7%) and the most frequent primary location was oral cavity (78.7%). The most frequent clinical stage was (CS) II (37.6%), the most histological grade was G2 (68.5%). Positive tumors for p16 were found in 10.1% (including 4 from the 9 oropharynx cases) and median Ki67 was 30%. Median follow-up was 16.3 months and 81 cases died. Staining of p16 was associated with oropharynx primary (44.4% vs 8.3%, p<0.001) and more frequent higher Ki67 tumor (88.9% vs 53%, p=0.004), but not to TIL (p=0.44), CD3 density (p=0.6), CD8 density (p=0.3) nor CD8/CD3 (p=0.13). Higher TIL was associated with higher Ki67 tumor (71.2% vs 48.8%, p=0.026), as well as with CD8 and CD3 density (Cohen's Kappa score= 0.698, p<0.001). Survival analysis found that CS III-IV were associated with shorter survival at 5-y (57.6 vs 63.4%, p=0.038). Higher TIL tends to be associated with longer survival (60 vs 51.2%, p=0.18). Neither p16 staining (p=0.679), ki67 (p=0.304) nor CD8/CD3 ratio (p=0.83) were associated with survival.

Conclusions

The presence of PVH surrogate marker, p16 is not frequent among Peruvian HNSCC, but associated with oropharynx location. Higher Ki67 was associated with both p16 and high TIL levels.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

FONDECYT 096-2017.

Disclosure

All authors have declared no conflicts of interest.

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