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ePoster Display

1106P - Phase I study of hepatic intralesional rose bengal disodium (PV10), an autolytic immunotherapy, in metastatic neuroendocrine neoplasms

Date

16 Sep 2021

Session

ePoster Display

Topics

Clinical Research;  Rare Cancers

Tumour Site

Neuroendocrine Neoplasms

Presenters

Timothy Price

Citation

Annals of Oncology (2021) 32 (suppl_5): S906-S920. 10.1016/annonc/annonc678

Authors

T.J. Price1, G. Cehic2, E. Wachter3, R. Sebben2, J. Reid4, A.M.I. Alawawdeh1, M. McGregor1, I. Kirkwood5, D. Rodrigues3, S. Neuhaus6, G. Maddern4

Author affiliations

  • 1 Medical Oncology, The Queen Elizabeth Hospital, 5011 - Woodville/AU
  • 2 Sami, The Queen Elizabeth Hospital, 5011 - Woodville/AU
  • 3 Development, Provectus Pharmaceuticals, 37931 - Knoxville/US
  • 4 Surgery, The Queen Elizabeth Hospital, 5011 - Woodville/AU
  • 5 Sami, Royal Adelaide Hospital, 5011 - Woodville/AU
  • 6 Surgery, University of Adelaide, 5000 - Adelaide/AU

Resources

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Abstract 1106P

Background

Metastatic neuroendocrine neoplasms (mNEN) require new treatment options. We investigated intralesional (IL) PV-10, an autolytic immunotherapy that may elicit a disease-specific functional adaptive immune response.

Methods

Phase 1 study evaluating safety, tolerability and impact on symptoms and biochemical markers resulting from IL PV-10 administered percutaneously to hepatic lesions in patients (pts) with progressive mNEN following standard therapy. Injected lesion(s) had to be 1.0 to 3.9 cm in longest diameter. Cohort 1 (n = 6): PV-10 IL single lesion per treatment cycle, Cohort 2 (n = 6): injections to multiple lesions per treatment cycle if suitable. Pts could receive further PV-10 ≥6 weeks after prior injection. The primary endpoint was safety. Secondary endpoints ORR assessed by contrast enhanced CT (RECIST 1.1) and [68Ga] Ga -DOTATATE PET, biochemical response (CgA) and patient-reported outcome (EORTC QLQ-C30 and GI.NET21).

Results

Twelve pts were enrolled, 50% male, median age 66 yrs (range 47-79). Primary sites: 7 small bowel, 2 pancreas, 1 caecal, 2 unknown; grade: Gd1 = 5, Gd2 = 7. All pts had received prior SSA and PRRT with progressive disease prior to enrolment. Median CgA was 1585 (range 35-10370). One lesion was injected per procedure for all 12 pts; none were suitable for multiple injections. Median dose per cycle (ml) was 1.9 (range 0.5-8.7). PV-10 treatment cycles; 4 n=1, 2 n=3, 1 n=8. Toxicity; Grade (gd) 1/2 transient post-procedure pain reported 9 pts, Photosensitivity rash gd 3 one patient; gd 1 elevation transaminases 2 pts, gd 3 in one, resolving by day 7. Gd 1/2 Carcinoid flare or increased flushing occurred in 6 pt. Gd 1/2 chromaturia was noted in 4 pts. ORR in injected lesions was 42%; overall disease control was 84%. CgA remained stable in 10 pts. Med PFS 9.2 months; OS 22.5 months. Estimated 3 yr PFS and OS is 27% & 49% respectively. HRQOL scores were stable or improved in 8 of 11 patients at mth1, and maintained at mth3 in 6 of 10 patients.

Conclusions

PV-10 elicited no safety concerns and multiple cycles were delivered safely in suitable patients. Encouraging evidence of local and systemic disease control with improvement in HRQoL was noted in a heavily pre-treated, progressing mNEN population.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

Provectus.

Funding

Provectus.

Disclosure

E. Wachter: Financial Interests, Personal, Full or part-time Employment: Provectus. D. Rodrigues: Financial Interests, Personal, Full or part-time Employment: Provectus. G. Maddern: Financial Interests, Institutional, Research Grant: Provectus. All other authors have declared no conflicts of interest.

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