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ePoster Display

294P - Cell-free DNA (cfDNA) workflow for the risk definition of dose-limiting and recurrent neutropenia in patients treated with first-line endocrine therapy (ET) and cyclin-dependent kinase 4/6 inhibitors (CDK4/6i) for metastatic breast cancer (MBC)

Date

16 Sep 2021

Session

ePoster Display

Topics

Management of Systemic Therapy Toxicities;  Targeted Therapy;  Translational Research;  Supportive Care and Symptom Management

Tumour Site

Breast Cancer

Presenters

Lorenza Palmero

Citation

Annals of Oncology (2021) 32 (suppl_5): S457-S515. 10.1016/annonc/annonc689

Authors

L. Palmero1, R. Mazzeo1, S. Buriolla1, L. Bortot1, A. Franzoni2, A. Michelotti1, G. Turra1, M. Zilli1, E.C. Stefani1, L. Allegri2, P. Di Nardo3, R. Roncato4, M. Bonotto5, E. Cecchin4, B. Belletti6, G. Toffoli4, G. Baldassarre6, G. Damante2, L. Gerratana1, F. Puglisi3

Author affiliations

  • 1 Department Of Medicine (dame), University of Udine - Dipartimento di Area medica - DAME, 33100 - Udine/IT
  • 2 Institute Of Human Genetics, Azienda Sanitaria Universitaria Friuli Centrale (ASUFC) Udine, 33100 - Udine/IT
  • 3 Department Of Medical Oncology, Centro di Riferimento Oncologico - CRO IRCCS, 33081 - Aviano/IT
  • 4 Clinical And Experimental Pharmacology, Centro di Riferimento Oncologico Aviano - IRCCS - Servizio Sanitario Regionale FVG, 33081 - Aviano/IT
  • 5 Medical Oncology Department, Azienda Sanitaria Universitaria Integrata di Udine - Ospedale Santa Maria della Misericordia, 33100 - Udine/IT
  • 6 Molecular Oncology Unit, Centro di Riferimento Oncologico, CRO Aviano National Cancer Institute, IRCCS, 33081 - Aviano/IT

Resources

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Abstract 294P

Background

Neutropenia (NP) is the main dose limiting toxicity of CDK4/6i. The present study aimed to evaluate the feasibility of a cfDNA-based workflow as a novel tool for assessing the risk of treatment-induced recurrent-neutropenia (rec-NP) and NP-induced CDK4/6i dose reduction (DR) in patients (pts) treated for luminal MBC.

Methods

A prospective cohort of 83 luminal MBC pts treated with first line ET and CDK4/6i in the CRO-2018-56 multicenter study was analyzed. cfDNA was characterized through droplet digital PCR (ddPCR) based on different ACTB DNA fragments lengths: short (s), medium (sl) and long (l). Blood samples were collected before treatment start (BL) and at the first clinical evaluation after 3 months (E1). Associations between clinical characteristics, cfDNA, rec-NP (≥3 NP events) and DR were explored through Kruskal Wallis.

Results

NP was higher than G2 in 46 out of 83 pts (55%). Overall, 29 pts (35%) developed rec-NP and 12 pts (26%) reduced CDK4/6i dose after NG3-G4. Notably, BL neutrophils count (Neu), WBC and lactate dehydrogenase (LDH) were significantly lower in pts that developed rec-NP (respectively p=0.0009, p=0.0008 and p=0.0375), while no significant associations with DR were observed. De novo metastatic pts had a lower risk of DR (p=0.0304) while pattern of metastasis was not associated with significant DR. BL ACTB_m was higher in pts that experienced DR (p=0.0096) while no associations emerged between ACTB_m and rec-NP. BL neu and ACTB_m were then combined to describe 4 distinct risk subgroups after dichotomization at the median value. Although pts with high ACTB_m and high neu did not experience any DRs, no significant differences were observed among subgroups (p=0.577).

Conclusions

The present study suggested that cfDNA can be used not only as a tumor-related biomarker in MBC, but also as a tool to assess the risk of drug-related adverse events, such as CDK4/6i-induced dose-limiting neutropenia. Additional investigations are planned to further refine the concept.

Clinical trial identification

CRO-2018-56.

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Ricerca Finalizzata Italian Ministry of Health Ricerca Finalizzata Italian Ministry of Health.

Disclosure

L. Gerratana: Financial Interests, Personal, Advisory Role: Lilly; Financial Interests, Personal, Advisory Role: Novartis; Non-Financial Interests, Personal, Other, Travel, Accommodations, Expenses: Menarini Silicon Biosystems. F. Puglisi: Financial Interests, Advisory Role: Roche; Financial Interests, Advisory Role: Amgen; Financial Interests, Advisory Role: Lilly; Financial Interests, Advisory Role: Novartis; Financial Interests, Advisory Role: Pfizer; Financial Interests, Advisory Role: Elisai; Non-Financial Interests, Other, Travel, Accommodations, Expenses: Celgene; Non-Financial Interests, Other, Travel, Accommodations, Expenses: Roche; Financial Interests, Other, Honoraria: Roche; Financial Interests, Other, Honoraria: MSD; Financial Interests, Other, Honoraria: AstraZeneca; Financial Interests, Other, Honoraria: Novartis; Financial Interests, Other, Honoraria: Lilly; Financial Interests, Other, Honoraria: Pfizer; Financial Interests, Other, Honoraria: Pierre Fabre; Financial Interests, Other, Honoraria: Daiichi Sankyo; Non-Financial Interests, Funding: Eisai; Non-Financial Interests, Funding: AstraZeneca; Non-Financial Interests, Funding: Roche. All other authors have declared no conflicts of interest.

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