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ePoster Display

1806P - Analysis of germ-line mutations in non-syndromic malignancies with concurrent pathogenic variants in BRCA1, BRCA2, PALB2, ATM, CHEK2, and RAD51C/D

Date

16 Sep 2021

Session

ePoster Display

Topics

Targeted Therapy

Tumour Site

Presenters

Emily Miller

Citation

Annals of Oncology (2021) 32 (suppl_5): S1227-S1236. 10.1016/annonc/annonc681

Authors

E.M. Miller1, J. Mullins2, A. Jain1, A. Chaudhry3, D. Mi1, R. Usman1, T. Jones1, Y. Baca4, J. Xiu5, W..M. Korn6, S. Cummings7, G. Vidal8

Author affiliations

  • 1 Oncology, University of Tennessee Health Sciences Center, TN 38103 - Memphis/US
  • 2 Oncology, Texas Oncology-San Antonio Northeast, 78217 - San Antonio/US
  • 3 Hematology Oncology, The university of Illinois College of Medicine, 60612 - Chicago/US
  • 4 2clinical And Translational Research, Medical Affairs, Caris Life Sciences, 85040 - Phoenix/US
  • 5 Clinical And Translational Research, CARIS Life Sciences - Arizona Office, 85054 - Phoenix/US
  • 6 Clinical And Translational Research, CARIS Life Sciences, 85054 - Phoenix/US
  • 7 Oncology Medical Affairs, Myriad Genetics, 84108 - Salt Lake City/US
  • 8 Medical Oncology, West Cancer Center, 38138 - Germantown/US

Resources

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Abstract 1806P

Background

Roughly 10% of breast cancers have an identifiable germline pathogenic variant, the most commonly being in the BRCA1/2 genes. Alterations in other genes, such as PALB2, ATM, CHK2, and RAD51C/D are also associated with increased risk of breast cancer when mutated. Pathogenic variants in these genes are also associated with other cancers. An increasing number of somatic mutations in synthetic lethal genes have been noted in “non-syndromic tumor” types; however, the clinical significance is unknown. We attempted to assess the rate of germline mutations in non-syndromic malignancies with concurrent somatic pathogenic mutations.

Methods

Patients with somatic pathogenic variants in BRCA1/2, PALB2, ATM, CHEK2, or RAD51C/D were identified via database review. Those with breast, ovarian, pancreatic, and prostate were excluded from the analysis. A chart review gathered clinical information and germline testing results. If no germline testing was completed, patients were contacted for single site germline testing by Myriad Genetics, Inc.

Results

Of the 247 patients with somatic mutations, 65 patients had existing germline testing or consented to single site germline variant analysis. This included 16 Black, 1 Hispanic, and 47 white patients. Colon adenocarcinoma was the most prevalent tumor type (40%), followed by NSCLC (19%), gastric adenocarcinoma (7%), bladder cancer (7%), and melanoma (6%). Of the 65 patients analyzed, 20 (13.6%) had germline variants in the corresponding target genes. The germline mutational distribution consisted of 30% BRCA1, 25% BRCA2, 25% ATM, 15% CHEK2, 5% PALB2, and no RAD51C/D. In comparing germline versus somatic variants, similar frequency was seen in BRCA1 (10.2% vs 12.1% respectively), PALB2 (1.7% vs 3.3%), and CHEK2 (5.1% vs 5.6%). However, BRCA2 and ATM variants were noted at higher frequency in the somatic genome, 33.3% BRCA2 and 29.8% ATM, compared with germline variants, 8.5%, and 8.5% respectively.

Conclusions

This small study demonstrates the relative frequency of germline BRCA1/2, PALB2, ATM, and CHEK2 in non-syndromic tumor types. The correlation between germline and somatic variants was most frequently noted for BRCA1, PALB2, and CHEK2.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

University of Tennessee Health Sciences Center.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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