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ePoster Display

1090TiP - A phase II, open label, multicenter study to investigate the efficacy and safety of domatinostat in combination with avelumab in patients with treatment-naïve metastatic Merkel cell carcinoma: The MERKLIN 1 study

Date

16 Sep 2021

Session

ePoster Display

Topics

Tumour Immunology;  Rare Cancers

Tumour Site

Basal Cell and Squamous Cell Cancers of the Skin

Presenters

Paolo Ascierto

Citation

Annals of Oncology (2021) 32 (suppl_5): S867-S905. 10.1016/annonc/annonc706

Authors

P.A. Ascierto1, A.C.J. van Akkooi2, P. Nghiem3, F. Hermann4, J. Henneberg5, J.C. Becker6

Author affiliations

  • 1 Melanoma, Cancer Immunotherapy & Developmental Therapeutics, Istituto Nazionale Tumori - IRCCS - Fondazione Pascale, 80131 - Napoli/IT
  • 2 Surgical Oncology Dept, NKI-AVL - Netherlands Cancer Institute/Antoni van Leeuwenhoek Hospital, 1066 CX - Amsterdam/NL
  • 3 Dermatology, UW Medical Center at Lake Union, 98109 - Seattle/US
  • 4 Clinical Development Department, 4SC AG, 82152 - Martinsried (Planegg)/DE
  • 5 Clin Ops, 4SC AG, 82152 - Planegg-Martinsried/DE
  • 6 Translational Skin Cancer Research, University Hospital Essen Westdeutsches Tumorzentrum, 45147 - Essen/DE

Resources

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Abstract 1090TiP

Background

Merkel cell carcinoma (MCC) is a rare and highly aggressive human skin cancer often caused by the Merkel cell polyomavirus or extended exposure to sunlight. Since the approvals of avelumab globally and subsequently pembrolizumab (US only), anti-PD-(L)1 antibody therapies have become standard of care for MCC patients in recent years. Despite these successes, a significant proportion of MCC patients do not respond or relapse on such monotherapy and have an unmet medical need for a more effective first-line therapy. Preclinical data suggest that the small molecule selective class I histone deacetylase inhibitor (HDACi) domatinostat addresses critical, well-described escape mechanisms of MCC. These escape mechanisms include the epigenetic downregulation of the antigen processing and presentation machinery leading to an insufficient recognition of tumor cells by the immune system. Treatment with domatinostat is hypothesized to favorably modulate the tumor environment and synergize with anti-PD-L1 therapy for a higher response rate, deeper responses, and longer duration of response.

Trial design

MERKLIN 1 (NCT04874831) is a phase II, multicenter, single arm clinical trial of the orally administered HDACi domatinostat in combination with the anti-PD-L1 antibody avelumab for patients with treatment-naïve metastatic MCC. A total of 100 patients will be enrolled in up to 40 clinical study sites initially in Europe. The primary objective is to evaluate the clinical efficacy of the combination in treatment-naïve metastatic or distally recurrent MCC patients as determined by the objective response rate, defined as the percentage of patients having a confirmed complete response or partial response according to Response Evaluation Criteria In Solid Tumors version 1.1 (RECIST v1.1) by independent review. Secondary objectives include additional efficacy assessments, safety, health related quality of life and pharmacokinetics of domatinostat and avelumab. Correlative aims include evaluating biomarkers for association with clinical benefit.

Clinical trial identification

4SC-202-4-2019 (Protocol Number), EudraCT 2019-003575-19.

Editorial acknowledgement

Legal entity responsible for the study

4SC AG, collaborator Merck KG.

Funding

4SC AG.

Disclosure

F. Hermann: Financial Interests, Personal, Full or part-time Employment: 4SC AG. J. Henneberg: Financial Interests, Personal, Full or part-time Employment: 4SC AG. All other authors have declared no conflicts of interest.

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