Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Lunch and Poster Display session

19P - HER2-low prognosis in early breast cancer: A CANTO based study (LILAC)

Date

16 May 2024

Session

Lunch and Poster Display session

Presenters

Lauriane DURAND

Citation

Annals of Oncology (2024) 9 (suppl_4): 1-34. 10.1016/esmoop/esmoop103010

Authors

L. DURAND1, F. Cherifi2, G. Emile2, C. Blanc-Fournier2, A. Morel2, A.C. Johnson2, P. Rottier2, I. Hrab2, P. Desbois2, C. Boscher2, C. Levy3, C. Dubot2, K. Gunzer4, B. Bimont2, C. Segura-Djezzar2, D. allouache2, J. Lequesne3, F. Christy2, A. Da Silva5

Author affiliations

  • 1 Centre Francois Baclesse, Caen, Cedex 5/FR
  • 2 Centre Francois Baclesse, Caen, Cedex/FR
  • 3 Centre Francois Baclesse, 14076 - Caen/FR
  • 4 Centre Francois Baclesse, 14000 - Caen, Cedex/FR
  • 5 Chu De Caen Normandie - Hôpital Clemenceau (CHR), Caen, Cedex/FR

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 19P

Background

As new therapeutic strategies in breast cancer (BC) emerge, human epidermal growth factor receptor 2 (HER2) low status has recently proved to have clinical value. There is still limited data available on this subgroup particularly in early settings. We aimed to evaluate the characteristics and prognosis of HER2 low status in patients with early BC.

Methods

We conducted a retrospective observational multicentric study using data from the French CANTO cohort (NCT01993498). Our primary objective was to assess invasive breast cancer-free survival (IBCFS) of HER low compared to HER2 0 BC. Secondary objectives were to evaluate invasive disease-free survival (IDFS), relapse-free interval (RFI) and overall survival (OS). Univariate and multivariate Cox regressions were performed.

Results

We included 9,956 patients with 5,802 HER2 0, 2,513 HER2 1+ and 1,241 HER2 2+ non-amplified (NA) patients corresponding to 9,980 tumours in total. Data were analysed at 100 months and the median follow-up duration was 73 months at this time. HER2 2+ NA tumours were significantly more likely to be larger than 20mm (p < 0.001), to be grade 3 (p < 0.001), to have a ki67 ≥ 20% (p < 0.001) and to be treated by chemotherapy (p < 0.001) compared to HER2 0 and HER2 1+ tumours. HER2 0 tumours were significantly less likely to be hormone receptor (HR) positive (p < 0.001), to have received hormonotherapy (p < 0.001) and were significantly more likely to be lobular tumours (p < 0.001), compared to HER2 1+ and 2+ NA. When we performed the analysis, no significant difference in IBCFS was observed between HER2 0, 1+, and 2+ NA (84.9%, 84.7% and 83.9% respectively, p=0,67). In the multivariate Cox model, HER2 1+ seemed to be associated with a worse IBCFS, but the difference was not significant (p = 0,036). There was no significant association between HER2 score and IDFS, RFI or OS.

Conclusions

We found that in early BC, although there was a significant difference in clinical and histological factors at diagnosis with HER2 2+ NA tumours being more aggressive, there was no significant difference in survival outcomes between HER2 negative and low status. Physiopathological studies and longer follow-up are needed.

Legal entity responsible for the study

Centre François Baclesse.

Funding

Centre François Baclesse.

Disclosure

G. Emile: Non-Financial Interests, Invited Speaker: AstraZeneca, Novartis, Gilead, Seagen, Lilly, Pfizer, Daiichi Sankyo. A. Da Silva: Non-Financial Interests, Invited Speaker: Leopharma, Novartis. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.