Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster Display session

513P - Identification of genetic profile using ctDNA in multiple myeloma

Date

07 Dec 2024

Session

Poster Display session

Presenters

Jin Ju Kim

Citation

Annals of Oncology (2024) 35 (suppl_4): S1580-S1594. 10.1016/annonc/annonc1694

Authors

J.J. Kim1, S. Shin2, S. Lim3

Author affiliations

  • 1 Laboratory Medicine, Yongin Severance Hospital, Yonsei University College of Medicine, 16995 - Yongin/KR
  • 2 Laboratory Medicine, Severance Hospital - Yonsei University College of Medicine, 03722 - Seoul/KR
  • 3 Graduate School Of Medical Science, Yonsei University College of Medicine, 120-752 - Seoul/KR

Resources

This content is available to ESMO members and event participants.

Abstract 513P

Background

Multiple myeloma (MM) is a hematologic malignancy characterized by accumulation of monoclonal neoplastic plasma cells. Despite the availability of new therapies, large number of patients still relapse. The increasing use of next generation sequencing (NGS) has shown that the mutation profile is related to the impact the patients’ outcome. However, due to the spatial genetic heterogeneity in the bone marrow (BM) compartment of MM patients, genetic assessment of a single bone marrow sample can lead incomplete interpretation and may not be able to provide enough information. Liquid biopsy is a method that analyzing genetic tumor profile of circulating tumor DNA (ctDNA) in the blood. ctDNA is expected to originate from tumor cells and blood is a cocktail of ctDNAs that can overcome the spatial genetic heterogeneity in MM patients. Here, we performed targeted-capture sequencing using bone marrow (BM) plasma cells and ctDNA of 40 MM cases.

Methods

A total of 40 MM patients were enrolled in a single center. DNA from CD138+ cells in BM were subjected to targeted-capture sequencing for 112genes and for 31 genes, respectively. We used a positional indexing sequencing (PiSeq) analysis method with high depth sequencing to increase the detection capability of NGS to below 1% of variant allele frequency (VAF).

Results

Mutations are detected in most of the patients, 94.4% (34/36) of BM samples and 78.9% (30/38) 0f ctDNAs. We detected 74 and 22 recurrently mutated genes in BM and ctDNA, respectively. We identified 24 and 13 driver genes in BM and ctDNA, respectively including KRAS, NRAS, BRAF and TP53. Clonal hematopoiesis (CH) associated mutations, particularly DNMT3A were frequently observed, not only in ctDNA but also in BM.

Conclusions

Our results indicate that both BM and ctDNA can detect mutations, but considering the VAF of the mutations, using CD138+ cells in BM might be preferred for confirming the genetic profile. However, it is noteworthy that even with a small panel targeting 31 genes in ctDNA, a significant number of driver gene mutations were identified. Further research on the correlation between ctDNA profiling and patient prognosis, especially regarding CH gene mutations, is needed.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

National Research Foundation of Korea.

Disclosure

J.J. Kim: Non-Financial Interests, Institutional, Funding: National Research Foundation of Korea. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.