Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster Display session

495P - DPyD screening “Opening the Pandora’s box in Indian population”: A pilot study

Date

07 Dec 2024

Session

Poster Display session

Presenters

S V Saju

Citation

Annals of Oncology (2024) 35 (suppl_4): S1580-S1594. 10.1016/annonc/annonc1694

Authors

H.S. Raju1, K.K. Rathnam1, S.V. Saju1, S. Dhanishka2

Author affiliations

  • 1 Medical Oncology Department, Meenakshi Mission Hospital and Research Centre, 625107 - Madurai/IN
  • 2 Department Of Medical Oncology, Meenakshi Mission Hospital and Research Centre, 625107 - Madurai/IN

Resources

This content is available to ESMO members and event participants.

Abstract 495P

Background

Mutations of DPyD can result in severe adverse reactions and even death in patients receiving fluoropyrimidines, a common chemotherapeutic agent. The spectrum of mutations and their clinical significance in Indians is not well characterized to date; resulting in unexpected toxicity in many patients.

Methods

To describe the spectrum of DPyD mutations in the screened population. To find causal associations between mutations and possible Treatment Related Adverse Events (TRAE) METHODOLOGY A Prospective analysis of all patients tested for DPyD deficiency between 1st January 2023 to 30th July2024, for a period of 4 chemo cycles. The incidence of TRAEs were noted and correlated with DPyD screening result.

Results

Of the 169 cases screened, 151 were analysed; of whom toxicity (any grade) was seen in 52(34.3%) patients [Grade III/IV toxicity-19 (12.5%), death-5 (Three in DPD wild type and two in the poor metaboliser mutation group)]. Nineteen required hospitalisation. Pre-emptive testing was done in 135 and for 16 after toxicity. Mutations were detected in 99 cases (65.5%); of which 34(22.5%) patients had TRAEs - any grade; and 12 (7.9%) - grade III/IV - including 2 deaths; both were harbouring poor metaboliser mutation. In the remaining 52 (34.4%) wild-type patients, 18 (11.9%) had toxicity (any grade); and 7 (4.6%) - grade III/IV- including 3 deaths. The most common mutation was c. 85T>C DPYD*9A p.C29R (69 out of 99; 69.6%) followed by c.2194G>A DPYD*6 p.V732I (14 out of 99; 14.4%). Among *9A mutatnts, 23 out 69 (33.33%) had any grade toxicity and 8 out of 69 (11.6%) had grade III/IV toxicity; and among *6 mutants, 7 out of 14 (50%) had any grade toxicity and 2 out of 14 (14.2%) had grade III/IV toxicity; which did not correlate with CPIC recommendations except in 2 cases. Both the patients who had non-functional mutations succumbed.

Conclusions

The spectrum of DPyD mutations in our study seemed different from the Dutch and CPIC databases. The toxicity did not correlate with CPIC recommendations, needing an entirely different dose modification guideline for Indian population.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.