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Poster Display session

357P - Comprehensive genomic profiling and treatment outcomes in mCRPC: Focusing on homologous recombination repair mutations

Date

07 Dec 2024

Session

Poster Display session

Presenters

Shinro Hata

Citation

Annals of Oncology (2024) 35 (suppl_4): S1531-S1543. 10.1016/annonc/annonc1690

Authors

S. Hata, H. Fujinami, M. Shinohara, S. Sejiyama, T. Inoue, T. Shin

Author affiliations

  • Urology, Oita University Faculty of Medicine, 879-5593 - Yufu/JP

Resources

This content is available to ESMO members and event participants.

Abstract 357P

Background

Homologous recombination repair gene mutations (HRRm) are found in approximately 25% of metastatic castration-resistant prostate cancer (mCRPC) cases. BRCA gene mutations, in particular, are associated with a more aggressive clinical course. The PROfound trial demonstrated the efficacy of PARP inhibitors in mCRPC with BRCA gene mutations. However, the effectiveness of androgen receptor signaling inhibitors (ARSIs) and chemotherapy in HRRm cases remains controversial. This study investigated the efficacy and clinical outcomes of ARSIs and taxane-based chemotherapy in mCRPC patients with HRRm.

Methods

We included mCRPC patients who underwent comprehensive genomic profiling (CGP) between March 2021 and July 2024. Patients were stratified based on the presence or absence of HRRm. The primary endpoint was PSA50/PSA90 response (≥50% or ≥90% reduction from baseline) to first-line ARSI treatment in both groups. Secondary endpoints included PSA30/PSA50 response (≥30% or ≥50% reduction) to docetaxel and overall survival from CGP submission and initial diagnosis in both groups.

Results

CGP was performed on 28 patients, with HRRm identified in 18 (64.3%) cases (ATM: 8, BRCA2: 5, BRCA1: 2, CDK12: 2, others: 3). The median age at diagnosis was 65 years, and median PSA was 44.5 ng/ml. Five patients each had undergone radical prostatectomy and radiation therapy with curative intent. There was no significant association between PSA50/PSA90 response to ARSIs and HRRm status (p = 0.68 and p = 0.21, respectively). Similarly, no significant association was found between PSA30/PSA50 response to docetaxel and HRRm status (p = 0.54 and p = 0.28, respectively). The median overall survival from CGP submission and initial diagnosis was 10.6 months and 170.1 months in the HRRm-positive group, and 12.3 months and 113.4 months in the HRRm-negative group, respectively. There was no evidence that OS differed based on HRRm status.

Conclusions

This study found no association between the treatment efficacy of ARSIs or taxane-based chemotherapy and HRRm status in mCRPC patients.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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