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E-Poster Display

734P - Kidney manifestations in patients with hereditary leiomyomatosis and renal cell cancer syndrome (LHRCC) in Spain

Date

17 Sep 2020

Session

E-Poster Display

Topics

Genetic Testing and Counselling;  Genetic and Genomic Testing

Tumour Site

Renal Cell Cancer

Presenters

Ana Beatriz Sanchez

Citation

Annals of Oncology (2020) 31 (suppl_4): S550-S550. 10.1016/annonc/annonc274

Authors

A.B. Sanchez1, A. Teule2, E. Lastra3, J. Balmana4, L. Robles5, J.D.D. García6, T. Ramón y Cajal7, G. Llort8, M. Fonfria9, M. Robledo10, A. Castillejo11, M. Duran12, A. Zuñiga13, L. Gómez14, E. Grau Garcés15, A. López4, M. Bosquet-Sanz16, V. Barberá11, J.L. Soto11

Author affiliations

  • 1 Medical Oncology, Hospital General Universitario de Elche, 03203 - Elche/ES
  • 2 2. hereditary Cancer Program, Catalan Institute of de Oncology, Barcelona/ES
  • 3 3. genetic Counselling Unit In Hereditary Cancer And Medical Oncology Department, Hospital Universitario de Burgos, Burgos/ES
  • 4 4. hereditary Cancer Genetics Group, Vhio. Medical Oncology Department, Vall d'Hebron University Hospital, 8035 - Barcelona/ES
  • 5 Unidad De Cáncer Familiar. Servicio De Oncología Médica, Hospital Universitario 12 de Octubre, Madrid/ES
  • 6 6. genetic Counselling Unit, Internal Medicine Dept, University Hospital of Príncipe de Asturias, Madrid/ES
  • 7 7. medical Oncology Deparment, Hospital Sant Pau i Santa Creu, Barcelona/ES
  • 8 8. hereditary Cancer Unit And Medical Oncology Department, Corporació Sanitaria Universitària Parc Taulí de Sabadell and Consorci Sanitari de Terrassa, Barcelona/ES
  • 9 9. cancer Genetic Counseling Unit, Medical Oncology, Hospital Provincial de Castellón, Castellón/ES
  • 10 10. hereditary Endocrine Cancer, Human Cancer Genetics Programme, Spanish National Cancer Centre (CNIO), Madrid/ES
  • 11 11. molecular Genetics Laboratory, Hospital General Universitario de Elche, Elche/ES
  • 12 12. instituto De Biología Y Genética Molecular, Universidad de Valladolid, Valladolid/ES
  • 13 13. genetic Clinic Unit, Hospital Politécnico y Universitario La Fe, Valencia/ES
  • 14 14. urology Dept., Hospital Universitario Sant Joan d'Alacant, Alicante/ES
  • 15 Hereditary Cancer Program, Catalan Insititute of de Oncology,Germans Trias i Pujol, Barcelona/ES
  • 16 17. urology Dept., Hospital Universitario General de Castellón, Castellón/ES

Resources

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Abstract 734P

Background

HLRCC is an autosomal dominant condition due to fumarate hydratase gene (FH) germline pathogenic variants. It is characterized by predisposition to skin and uterine leiomyomas, renal cysts (RCy) and renal cell cancer (RCC). So far, no phenotype/genotype correlations have been described. We aimed to characterize the genetic, clinical, epidemiological and pathological features of RCy and RCC in the largest Spanish series of FH variants carriers.

Methods

We performed a multicentre, observational, retrospective study of a cohort of 197 patients, 74 index, with genetic diagnosis of HLRCC. We analysed FH pathogenic variants, presence of RCy and RCC, diagnosis age, histology, stage, treatment, survival and known risk factors for RCC as arterial hypertension, tobacco exposure and obesity using the software R v3.6.0. The study was approved by the Hospital General Universitario de Elche Ethics Committee.

Results

We identified 27 FH pathogenic variants: 13 missense, 5 frameshift, 4 large deletions, 3 splice site, 2 nonsense. Of 153 patients with radiological records 57 presented RCy (37.3%), which were more frequent in missense variants carriers (p = 0.017). Tobacco exposure (p= 0.076), and obesity (p = 0.058) show a trend to RCy. There were 19 cases of RCC (10.9%) among 175 patients with confirmed clinical information, 11 males/8 females, 7 with RCy. The median age at diagnosis was 37 years, range 10-67. One patient presented 2 synchronous bilateral RCC. Histological patterns were 14 papillary (10 type 2), 4 clear cells, 2 unclassified carcinoma. No significant differences were found by type of variant or risk factors. Six were stage I, 2 stage II, 3 stage III, 4 stage IV, and 4 not available. The median overall survival (mOS) in stages III-IV was 2.9 years [CI 95% 2.4-3.4]. In stages I-II the mOS is not reached. Six patients with metastatic disease received anti-angiogenic treatments with mOS 34.9 months [CI 95%: 29.0-40.9].

Conclusions

RCy were more frequent in missense mutations carriers. The RCC frequency (10.9%) was lower than in other published cohorts. Being papillary type-2 the most frequent histology, other histological patterns do not exclude HLRCC. Anti-angiogenic treatment offered similar results of overall survival than in sporadic RCC.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

All authors have declared no conflicts of interest.

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