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Poster Display session

93P - MDX-124, a novel anti-ANXA1 antibody, has significant anti-cancer activity in preclinical models of osteosarcoma

Date

21 Mar 2025

Session

Poster Display session

Presenters

Fiona Dempsey

Citation

Annals of Oncology (2025) 10 (suppl_3): 1-30. 10.1016/esmoop/esmoop104375

Authors

F. Dempsey1, S. Crichton1, C. Parris2, R. Ibrahem2, H. Al-Ali2, C. Pepper3, A. Patel4

Author affiliations

  • 1 Research And Development, Medannex Ltd, EH3 9QA - Edinburgh/GB
  • 2 School Of Life Science, ARU - Anglia Ruskin University - Main Campus, CB1 1PT - Cambridge/GB
  • 3 Clinical And Experimental Medicine, Brighton and Sussex Medical School - University of Sussex, BN1 9PX - Brighton/GB
  • 4 Dept Of Oncology, St Jude Children's Research Hospital, 38105 - Memphis/US

Resources

This content is available to ESMO members and event participants.

Abstract 93P

Background

Osteosarcoma (OS) is a rare primary cancer of the bone characterised by its aggressive nature, strong invasiveness, rapid disease progression and high mortality rate. As patient outcomes have stagnated in the last 40 years, there remains a high unmet clinical need for novel targeted therapies to treat OS. Annexin-A1 (ANXA1), a 37 kDa phospholipid-binding protein, is overexpressed in several tumours, with high expression correlating with both poor patient outcomes and tumour growth and metastasis. Data from the St Jude-Washington University Children's Research Hospital Pediatric Cancer Genome Project (PCGP) identified a substantial fraction of OS with high ANXA1 expression suggesting it is a novel therapeutic target in OS. MDX-124 is a first-in-class humanised antibody targeting ANXA1 currently being evaluated in a phase I clinical trial (ATTAINMENT). Here we present data demonstrating efficacy of MDX-124 in preclinical OS models.

Methods

ANXA1 gene expression and protein levels in OS samples were assessed via RNA-seq and immunohistochemistry respectively. Imaging flow cytometry measured ANXA1 expression and cellular localisation in human OS cell lines. OS cell viability following incubation with MDX-124 alone or in combination with standard of care therapies (methotrexate, doxorubicin, cisplatin, ifosamide or etoposide) was assessed via MTT assay. The effect of MDX-124 on OS cell cycle progression was evaluated by measuring changes in DNA content using flow cytometry. A transwell assay was used to evaluate the impact of MDX-124 on OS cell migration.

Results

OS tissues and human OS cell lines displayed high levels of ANXA1 expression. Incubation of OS cell lines with MDX-124 significantly reduced cell growth via cell cycle arrest in a dose-dependent manner. MDX-124 also significantly reduced the migratory ability of OS cell lines and enhanced the anti-cancer activity of several standard of care OS therapies.

Conclusions

MDX-124 targets secreted ANXA1 preventing activation of FPR1/2 and reducing cancer progression. MDX-124 has demonstrated anti-tumour activity in several preclinical models of OS. Medannex plans to initiate a phase Ib study of MDX-124 in paediatric OS in late 2025.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

Medannex Limited.

Funding

Medannex Limited.

Disclosure

F. Dempsey, S. Crichton: Financial Interests, Personal, Full or part-time Employment: Medannex Limited. C. Parris, R. Ibrahem, H. Al-Ali, C. Pepper: Financial Interests, Institutional, Research Grant: Medannex Limited. All other authors have declared no conflicts of interest.

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