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Poster Display session

82P - Advancing precision oncology in soft tissue sarcomas: The role of iTRAC in metastatic risk stratification and treatment personalization

Date

21 Mar 2025

Session

Poster Display session

Presenters

Frederic Chibon

Citation

Annals of Oncology (2025) 10 (suppl_3): 1-30. 10.1016/esmoop/esmoop104375

Authors

F. Chibon1, A. Benhaddou2, G. Pérot1, P. Rochaix3, T. Valentin4, G. Ferron5

Author affiliations

  • 1 Oncosarc, CRCT - Centre de Recherches en Cancérologie de Toulouse, 31100 - Toulouse/FR
  • 2 R&d, Magic Genomix, 31100 - Toulouse/FR
  • 3 Pathology Dept., IUCT - Institut Universitaire du Cancer de Toulouse - Oncopole, 31059 - Toulouse/FR
  • 4 Medical Oncology Department, IUCT - Institut Universitaire du Cancer de Toulouse - Oncopole, 31059 - Toulouse/FR
  • 5 Oncosarc, IUCT - Institut Universitaire du Cancer de Toulouse - Oncopole, 31059 - Toulouse/FR

Resources

This content is available to ESMO members and event participants.

Abstract 82P

Background

Soft Tissue Sarcomas (STS), known for their extensive Genomic instability (GIN), often result in poor clinical outcomes. grading systems, such as FNCLCC, have limited prognostic accuracy in stratifying metastatic risk for STS patients. We introduce transcription-associated GIN indice (iTRAC) to improve prognostic precision and guide treatment decisions.

Methods

This study analyzed 226 STS tumor samples using RNA sequencing (RNAseq) to assess breakpoint (BP) distributions from fusion transcripts as a surrogate for GIN. We calculated iTRAC to quantify transcription--associated GIN. Kaplan-Meier survival analysis were used to evaluate prognostic relevance in patients receiving or not chemotherapy. Multivariate analyses were performed to evaluate the iTRAC compared to FNCLCC and CINSARC for metastatic risk stratification.

Results

STS patients with medium iTRAC levels had the poorest metastasis-free survival (MFS) compared to low and high iTRAC levels. Importantly, patients with low iTRAC have a poorer outcome when treated with chemotherapy than those untreated, but at the contrary patients with medium iTRAC and treated by chemotherapy have a better outcome, raising the question of the potential predictive value of iTRAC for adjuvant chemotherapy in STS patients. FNCLCC and CINSARC groups did not show significant difference in MFS between treated and not treated patients.

Conclusions

iTRAC is a novel biomarker for stratifying metastatic risk and guiding personalized treatment in STS. It outperforms systems like CINSARC and FNCLCC by distinguishing low- and medium-iTRAC groups, revealing distinct MFS between patients receiving or not chemotherapy. This enables better identification of patients who may benefit from chemotherapy and alternative options for those with poor responses. Prospective clinical trials are needed for validation and routine integration.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

INSERM.

Funding

INSERM, ICGC.

Disclosure

A. Benhaddou: Financial Interests, Personal and Institutional, Stocks/Shares: Magic Genomix. All other authors have declared no conflicts of interest.

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