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Poster Display session

128P - Real-world survival outcomes in MSS colorectal cancer(CRC) patients based on mesothelin(MSLN) expression

Date

27 Jun 2024

Session

Poster Display session

Presenters

Midhun Malla

Citation

Annals of Oncology (2024) 35 (suppl_1): S1-S74. 10.1016/annonc/annonc1477

Authors

M. Malla1, S.K. Deshmukh2, S. Wu2, T. Samec2, M. Akce3, D. Olevian4, R. Elnaili4, J. Xiu5, A. Farrell2, H.J. Lenz6, E. Lou7, S. Goel8, D. Spetzler9, R.M. Goldberg10, L. Hazlehurst10, B.F. El-Rayes11

Author affiliations

  • 1 West Virginia University Mary Babb Randolph Cancer Center, Morgantown/US
  • 2 Caris Life Sciences - Headquarters, Irving/US
  • 3 The University of Alabama at Birmingham, Birmingham/US
  • 4 WestVirginiaUniversity, Morgantown/US
  • 5 Caris Life Sciences, Phoenix/US
  • 6 University of Southern California Norris Comprehensive Cancer Center, Los Angeles/US
  • 7 Masonic Cancer Center - University of Minnesota, Minneapolis/US
  • 8 Rutgers Cancer Institute of New Jersey, New Brunswick/US
  • 9 Caris Life Sciences, New York/US
  • 10 West Virginia University, Morgantown/US
  • 11 University of Alabama at Birmingham, 35233 - Birmingham/US

Resources

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Abstract 128P

Background

MSLN overexpression ranges from 40-45% in CRC and is associated with aggressive clinicopathological features and worse survival. We analyzed the association of MSLN expression based on RNA sequencing with molecular features, tumor microenvironment, enrichment of cancer related pathways, and overall survival(OS) in CRC.

Methods

14,892 CRC samples were tested by NGS, WES, WTS (Caris Life Sciences, Phoenix, AZ). Tumors with MSLN-high(H) and MSLN-low(L) expression were classified by top and bottom quartile, respectively. Pathway enrichment was calculated through Gene Set Enrichment Analysis (GSEA). Chi-square and Mann-Whitney U tests with p-values adjusted for multiple comparisons (q<0.05) were used. Real world OS was extracted from insurance claims and calculated using Kaplan-Meier estimates from treatment start to last contact.

Results

MSLN-H tumors had enrichment of inflammatory, IFN-γ, IL2/STAT5, IFN-α responses, IL6/JAK/STAT3, TNFα and TGFβ signaling pathways (NES: 1.3-1.4, all FDR < 0.25). MSLN-H tumors had higher expression of immune-related genes TNF, TIGIT, VTCN1, IL1A, IL1B, CD274, PDCD1, PDCD1LG2, CTLA4, LAG3, HAVCR2, FOXP3 (FC: 1.1-1.8), but lower expression of IL12A (FC: 1.1) (all q<0.05). MSLN-H tumors had higher IHC-PD-L1 positivity (4.4% vs 2.0%, q<0.05). Median MSLN expression was not associated with post-5-FU-chemotherapy OS (MSLN- > Median vs -< Median: 37.0 vs 40.9 m, HR: 0.93, 95% CI 0.86 – 1.0, p=0.09) but numericaly improved OS post-Nivolumab (MSLN- > Median (n=37) vs -< Median (n=30): 13.0 vs 4.6 m; HR: 1.7, 95% CI 0.95 – 3.1, p=0.06). Quartile MSLN expression was associated with worse post-5-FU-chemotherapy OS (MSLN- top 25% vs - bottom 25%: 33.3 vs 40.5 m, HR: 0.87, 95% CI 0.77 – 0.97, p=0.01) but improved OS post-Nivolumab (MSLN-top 25% (n=19) vs - bottom 25% (n=11): 12.2 vs 3.9 m; HR: 4.8, 95% CI 1.5 – 14.9, p=0.003).

Conclusions

CRC patients with MSLN-H expression treated with chemotherapy have inferior prognosis compared to MSLN-L CRC. However, OS for MSLN-H MSS CRC patients who received immunotherapy was significantly improved compared to the latter. Our results support further investigation of MSLN as a predictive biomarker for immunotherapy in future prospective studies in MSS CRC patients.

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

M. Malla: Financial Interests, Personal, Advisory Board: Natera, Exelixis, AstraZeneca. S.K. Deshmukh, S. Wu, J. Xiu, A. Farrell, D. Spetzler: Financial Interests, Personal and Institutional, Stocks/Shares: Caris. All other authors have declared no conflicts of interest.

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