Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster Display session

51P - Phase II dose expansion study of DSP107, a first-in-class fusion protein targeting CD47 and 4-1BB, in combination with atezolizumab in patients with advanced MSS colorectal cancer

Date

27 Jun 2024

Session

Poster Display session

Presenters

Anwaar Saeed

Citation

Annals of Oncology (2024) 35 (suppl_1): S1-S74. 10.1016/annonc/annonc1477

Authors

A. Saeed1, J. Zhang2, B. Bashir3, A. Philipovskiy4, R. Chantre5, S. Amsili5, R. Tabakman5, Y. Shwartz5, A. Foley-Comer5, A. Jimeno6

Author affiliations

  • 1 University of Pittsburgh Medical Center Hillman Cancer Center, 15219 - Pittsburgh/US
  • 2 KUMC - University of Kansas Medical Center, Kansas City/US
  • 3 Sidney Kimmel Cancer Center - Thomas Jefferson University, Philadelphia/US
  • 4 Florida Cancer Specialists & Research Institute - Lake Mary Cancer Center, Lake Mary/US
  • 5 KAHR Bio, Modi'in/IL
  • 6 UCHealth Cancer Care - Anschutz Medical Campus - University of Colorado Cancer Center, Aurora/US

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 51P

Background

DSP107 is a fusion protein that combines elements of SIRPα and the 4-1BB ligand (4-1BBL). Its SIRPα component targets CD47 on tumor cells, effectively activating macrophages by neutralizing the "don't eat me” signal, while simultaneously anchoring trimeric 4-1BBL to the tumor, to co-stimulate 4-1BB on activated immune cells in the tumor microenvironment. This results in tumor-localized, conditional activation of innate and adaptive immune responses. Phase 1 data demonstrated an excellent safety profile with no red blood cells binding and no hematological, hepatic or other dose limiting toxicities (DLTs). Here we describe safety and efficacy data from phase 2 microsatellite stable (MSS) colorectal (CRC) expansion cohort treated with DSP107 plus atezolizumab.

Methods

Stage 3b/4 MSS-CRC patients who progressed following 2 lines of therapy including standard chemotherapy and/or targeted antibodies (n=27), were treated with weekly IV DSP107 infusions (10 mg/kg) and atezolizumab (1200 mg) Q3W during 3-week treatment cycles. The majority (67%) had liver metastases and BRAF/KRAS mutations. Study objectives were safety, tolerability and preliminary efficacy. Restaging imaging was performed every 2 months and evaluated by RECIST v1.1 criteria.

Results

DSP107 with atezolizumab was well tolerated with no DLTs. The most frequent TRAEs were infusion-related reactions (IRR; 44.4%), fatigue (18.5%), grade 1 anemia and eosinophilia (11.1% each). IRRs were managed during subsequent infusions by reducing the infusion rate and administering IV fluids. A median PFS of 20 weeks was observed in 21 evaluable patients. Disease control was demonstrated in 62% of evaluable patients (13/21) including two patients with deep (≥80% target lesion reduction) and durable (>15 months) confirmed objective responses and disappearance of pulmonary and hepatic metastases.

Conclusions

DSP107 demonstrated a distinct safety and efficacy profile. These phase 2 data suggest that the combination of DSP107 with PD(L)1 blockade has anti-tumor activity in MSS CRC including in patients with liver metastases and warrants further clinical investigation.

Clinical trial identification

NCT04440735.

Legal entity responsible for the study

KAHR Medical.

Funding

KAHR Medical.

Disclosure

R. Chantre, S. Amsili, R. Tabakman, Y. Shwartz, A. Foley-Comer: Financial Interests, Personal, Full or part-time Employment: KAHR Medical. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.