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Poster Display session

182P - A real-world, multicenter study of atezolizumab plus bevacizumab in viral and non-viral advanced hepatocellular carcinoma patients from Asia and Europe

Date

27 Jun 2024

Session

Poster Display session

Presenters

Federico Rossari

Citation

Annals of Oncology (2024) 35 (suppl_1): S75-S93. 10.1016/annonc/annonc1478

Authors

F. Rossari1, T. Tada2, S. Shimose3, M. Kudo4, C. Yoo5, J. Cheon6, F. Finkelmeier7, H.Y. Lim8, J. Presa9, G. Masi10, F. Bergamo11, E. Amadeo12, F. Vitiello12, S. Foti13, S. Camera13, M. PERSANO14, B. Stefanini15, M. Scartozzi16, M. Rimini12, A. Casadei Gardini12

Author affiliations

  • 1 IRCCS Ospedale San Raffaele, 20132 - Milan/IT
  • 2 Japanese Red Cross Society Himeji Hospital, Himeji/JP
  • 3 Kurume University Hospital, Kurume/JP
  • 4 Kindai University Faculty of Medicine, Osaka/JP
  • 5 Asan Medical Center - University of Ulsan, Seoul/KR
  • 6 Ulsan University Hospital, Ulsan/KR
  • 7 Goethe-University Frankfurt am Main - Campus Westend, Frankfurt am Main/DE
  • 8 Samsung Medical Center (SMC) - Sungkyunkwan University School of Medicine, Seoul/KR
  • 9 rás-os-Montes e Alto Douro Hospital Centre, Vila real/PT
  • 10 Unit of Medical Oncology 2, Azienda Ospedaliera Universitaria Pisana, Pisa/IT
  • 11 IOV - Istituto Oncologico Veneto IRCCS, Padova/IT
  • 12 IRCCS Ospedale San Raffaele, Milan/IT
  • 13 Vita-Salute San Raffaele University, IRCCS San Raffaele Scientific Institute Hospital, Milan/IT
  • 14 AOU di Cagliari - Ospedale Civile, Cagliari/IT
  • 15 Imperial College London - Hammersmith Campus, London/GB
  • 16 University Hospital and University of Cagliari, 9042 - Monserrato/IT

Resources

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Abstract 182P

Background

Etiology impact on immunotherapy response in advanced hepatocellular carcinoma (HCC) is being debated, with contrasting findings between early and recent post-hoc analyses of IMbrave-150 and metanalyses of clinical trials of PD-1/PD-L1 blockers. As a result, whether the I-line atezolizumab plus bevacizumab (A+B) is equally effective in viral and non-viral patients (pts) remains unclear.

Methods

We retrospectively analyzed data from 885 HCC pts treated with I-line A+B from multiple centers from Europe and Asia, 53.9% having viral and 46.1% non-viral etiology. Baseline clinical and laboratory characteristics were analyzed with uni- and multi-variate models to explore potential differences on overall survival (OS), time to progression (TTP), disease control rate (DCR) and to identify putative prognostic factors in etiology subgroups. Treatment toxicities and access to II-line treatments and outcomes were also compared between etiologies.

Results

No statistically significant differences were found in OS (mOS: viral 15.9mo; non-viral 16.3mo), TTP (mTTP: viral 8.3mo; non-viral 7.2mo), DCR (viral 78.1%; non-viral 80.8%) based on etiology. Independent prognostic factors of survival and progression were mainly shared between viral and non-viral etiologies, including alpha-fetoprotein, aspartate transaminase, neutrophil-to-lymphocyte ratio (NLR) and ALBI score. Exploratory analyses highlighted a possible stronger association of immunological factors, i.e. NLR and eosinophil count, to treatment outcomes in viral pts. The toxicity profile, the access to and type of II-line treatments and their outcome in terms of OS almost overlap in the two etiology subgroups.

Conclusions

A+B efficacy does not vary according to underlying etiology of HCC in a multicenter, real-world population, matching recent post-hoc findings from the IMbrave-150 trial. Preliminary analyses suggest that some prognostic factors differ between viral and non-viral pts, potentially due to biological and immunological differences. Prospective comparative trials stratifying by etiology are warranted to validate these findings and guide clinical practice.

Legal entity responsible for the study

The authors.

Funding

Has not received any funding.

Disclosure

A. Casadei Gardini: Financial Interests, Personal and Institutional, Advisory Role: MSD, Eisai, Bayer, Bristol Myers Squibb, AstraZeneca, GSK. All other authors have declared no conflicts of interest.

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