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ePoster Display

1215P - EGFR-mutated squamous cell lung cancer and its association with outcomes

Date

16 Sep 2021

Session

ePoster Display

Topics

Tumour Site

Non-Small Cell Lung Cancer

Presenters

Yang Xia

Citation

Annals of Oncology (2021) 32 (suppl_5): S949-S1039. 10.1016/annonc/annonc729

Authors

Y. Xia1, R. Jin1, L. Peng2, J. Shou3, J. Wang4, Y. Jin5, F. Liang6, J. Zhao7, M. Wu8, Q. Li1, B. Zhang1, X. Wu8, F. Lan1, L. Xia1, J. Yan8, Y. Shao8, J. Stebbing9, H. Shen1, W. Li1

Author affiliations

  • 1 Department Of Respiratory And Critical Care Medicine, The Second Affiliated Hospital of Zhejiang University School of Medicine, 310009 - Hangzhou/CN
  • 2 Department Of Radiotherapy, First Affiliated Hospital, School of Medicine, Hangzhou/CN
  • 3 Department Of Medical Oncology, Sir Run Run Shaw Hospital, College of Medicine, Zhejiang University, Hangzhou/CN
  • 4 Department Of Medical Oncology, Cancer Hospital of the University of Chinese Academy of Sciences, 310000 - Hangzhou/CN
  • 5 Department Of Radiation Oncology, Cancer Hospital of the University of Chinese Academy of Sciences, 310000 - Hangzhou/CN
  • 6 Department Of Biostatistics, Zhongshan Hospital, Fudan University, 200032 - Shanghai/CN
  • 7 Department Of Medical Oncology, The Second Affiliated Hospital of Zhejiang University School of Medicine, 310009 - Hangzhou/CN
  • 8 Translational Medicine Research Institute, Geneseeq Technology Inc., Toronto/CA
  • 9 Division Of Cancer, Department Of Surgery And Cancer, Imperial College London, London/GB

Resources

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Abstract 1215P

Background

The therapeutic efficacy of epidermal growth factor receptor tyrosine kinase inhibitors (EGFR-TKIs) in advanced EGFR-mutant lung squamous cell carcinoma (SCC) patients remains uncertain. Furthermore, the factors underlying the responsiveness have not been fully investigated. We therefore investigated the link between genomic profiles and EGFR-TKI efficacy.

Methods

We consecutively enrolled stage IV, EGFR-mutant-, and EGFR-TKI-treated patients with SCC. Patients with EGFR wild-type lung SCC and EGFR-mutant lung adenocarcinoma were consecutively enrolled as controls, and next-generation sequencing (NGS) was performed.

Results

In total, 28 EGFR-mutant lung SCC, 41 EGFR-mutant lung adenocarcinoma, and 40 EGFR wild-type lung SCC patients were included. Among the patients with EGFR mutations, shorter progression-free survival (PFS) was observed in SCC compared to adenocarcinoma (4.6 vs. 11.0 months, P<0.001). Comparison of the genomic profiles revealed that EGFR-mutant SCC patients had similar mutation characteristics to EGFR-mutant adenocarcinoma patients, but differed from those with EGFR wild-type SCC. Further exploration of EGFR-mutant SCC revealed that mutations in CREBBP (P=0.005), ZNF217 (P=0.016), and the Wnt (P=0.027) pathway were negatively associated with PFS. Mutations in GRM8 (P=0.025) were associated with improved PFS.

Conclusions

EGFR-mutant lung SCC has a worse prognosis than EGFR-mutant adenocarcinoma. Mutations in other genes, such as CREBBP, ZNF217, GRM8 or Wnt that had implications on PFS raise the possibility of understanding mechanisms of resistance to EGFR-TKI in lung SCC, which will aid identification of potential beneficial subgroups of patients with EGFR-mutant SCCs receiving EGFR-TKIs.

Clinical trial identification

Editorial acknowledgement

Legal entity responsible for the study

The authors.

Funding

Zhejiang Provincial Natural Science Foundation [LY20H010004]; National Natural Science Foundation of China [81870022].

Disclosure

M. Wu, X. Wu, J. Yan, Y. Shao: Financial Interests, Institutional, Full or part-time Employment: Geneseeq Technology Inc. All other authors have declared no conflicts of interest.

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