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Lunch and Poster Display session

222P - Poor response to cyclin dependent kinase 4/6 inhibition in metastatic inflammatory breast cancer

Date

16 May 2024

Session

Lunch and Poster Display session

Presenters

Azadeh Nasrazadani

Citation

Annals of Oncology (2024) 9 (suppl_4): 1-47. 10.1016/esmoop/esmoop103200

Authors

A. Nasrazadani1, R. Slack1, M. Kai2, B. Lim3, V. Valero4, D. Tripathy5, S. Saleem2, A. LUCCI6, W. Woodward2, R. Layman1

Author affiliations

  • 1 MD Anderson Cancer Center, Houston/US
  • 2 University of Texas MD Anderson Cancer Center, Houston/US
  • 3 The University of Texas MD Anderson Cancer Center - Breast Medical Oncology, Houston/US
  • 4 The M.D. Anderson Cancer Center, Houston/US
  • 5 MD Anderson Cancer Center, 77030 - Houston/US
  • 6 The University of Texas M. D. Anderson Cancer Center, Houston/US

Resources

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Abstract 222P

Background

Patients with inflammatory breast cancer (IBC) have a poor prognosis owing to the aggressive biology of the disease and relatively inferior response to standard of care therapies. Cyclin dependent kinase 4/6 inhibitors (CDKI) are utilized in the metastatic setting for hormone receptor-positive HER2-negative/low (HR+ HER2-) breast cancer. The outcomes of these patients treated with CDKI combined with hormone directed therapy are presented.

Methods

Patients with HR+ HER2- IBC for which CDKI was administered in the metastatic setting were identified from the IBC registry at MD Anderson Cancer Center (N= 58). Medians (95% confidence intervals) of overall survival (OS), progression free survival (PFS), and time on treatment (ToT) from the time of CDKI initiation are reported by Kaplan-Meier methods. Differences are tested by log-rank test.

Results

All patients (42 stage III, 16 de novo stage IV) received induction chemotherapy at diagnosis. Median OS, PFS, and ToT in the total cohort was 26 (16, 37), 7 (5, 10), and 7 (5, 10) months (mos), respectively. No differences were observed between pre and post-menopausal patients. OS, PFS, and ToT in initial diagnosis of Stage III (N=42) vs IV (N=16) patients was 19 (15, 34) vs 34 (21, NR), 7 (5, 14) vs 9 (6, NR), and 6 (5, 10) vs 9 (4, NR) mos, respectively (ns). Patients treated with abemaciclib (N=11), Palbociclib (N=36), and ribociclib (N=9) show PFS of 7 (3, NR), 6 (4, 10), 9 (7, NR) mos; and ToT of 6 (3, NR), 6 (4, 9), 9 (7, NR) mos. OS, PFS, and ToT in patients receiving CDKI in the fistr-line vs second-line metastatic setting were 27 (19, 44) vs 17 (12, 39), 7 (5, 15) vs 6 (3, NR), and 7 (5, 15) vs 6 (3, 20) mos, respectively (ns). Brain metastases were observed in 12/42 stage III patients.

Conclusions

Patients with metastatic HR+ HER2- IBC demonstrate a poor response to CDKI based therapy. Patients receiving CDKI in the first vs second-line setting demonstrated a relatively superior outcome. Overall, however, outcomes are significantly inferior to historical data compared to non-IBC. Small sample size precludes definitive conclusions and data should be interpreted cautiously.

Legal entity responsible for the study

A. Nasrazadani.

Funding

Has not received any funding.

Disclosure

A. Nasrazadani: Other, Personal, Invited Speaker: AstraZeneca; Other, Personal, Advisory Role: Novartis. All other authors have declared no conflicts of interest.

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