Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Lunch and Poster Display session

83P - Boron-doped alginate carbon-nanogel exhibits superior anti-metastatic effect on breast cancer by cell cycle arrest and actin dysfunction

Date

16 May 2024

Session

Lunch and Poster Display session

Presenters

Saumyadip Sarkar

Citation

Annals of Oncology (2024) 9 (suppl_4): 1-34. 10.1016/esmoop/esmoop103010

Authors

S. Sarkar1, B. Unnikrishnan1, C. Wang1, C. Lin1, A. Anand2, R. Shih1, L.E. Hean1, C. Huang1

Author affiliations

  • 1 National Taiwan Ocean University, Keelung City/TW
  • 2 Chang Gung University, Taoyuan City/TW

Resources

Login to get immediate access to this content.

If you do not have an ESMO account, please create one for free.

Abstract 83P

Background

Metastasis is widely recognized as the primary contributor to mortality in cancer-related cases; however, the precise mechanisms responsible for this phenomenon have yet to be fully elucidated. The potential of natural compounds, particularly polysaccharides and polyphenols, as medicines with anti-tumor and anti-metastasis effects has attracted increasing attention in recent years. Therapeutic efficacy may be improved by the use of nanomaterials that can be controlled and used as delivery systems. In our findings, we employed boron-doped alginate carbon nanogels Bor(5)/Alg-240-CNGs that demonstrate a higher degree of efficacy in suppressing the migration and invasion of cancer cells in triple-negative breast cancer (TNBC).

Methods

Migration assays, invasion assays, cell attachment assays, microcolony analyses, and cell cycle analyses are used to assess the anti-metastatic properties of Bor(5)/Alg-240-CNGs in vitro. Different types of breast cancer cells and normal cells were tested for their sensitivity to Bor(5)/Alg-240-CNGs in order to learn more about their biocompatibility. The DCFH-DA assay and apoptosis analysis were then performed to examine anti-cancer activity further. In vivo, these nanomaterials were used to cure 4T1-induced tumors in BALB/c mice. Finally, proteomics provided much-needed insight into how exactly these nanomaterials produce their anti-metastatic actions.

Results

In our findings, we employed Bor(5)/Alg-240-CNGs that demonstrate a higher degree of efficacy in suppressing the migration and invasion of cancer cells in TNBC and also TNBC-dependent cell cytotoxicity due to ROS induced apoptosis. Further in vitro mechanisms show Bor(5)/Alg-240-CNGs induce cell cycle arrest and F-actin disorganization which is an essential factor for paralyzing migration. In the in vivo study, therapy with Bor(5)/Alg-240-CNGs resulted in an 86.16 % decrease in the number of metastatic lung nodules compared to the untreated group. The overall mechanism was further validated using proteomics.

Conclusions

Finally, Bor(5)/Alg-240-CNGs have the potential to be a cost-effective, inventive, and unique nanodrug with many targets for cancer treatment.

Legal entity responsible for the study

The authors.

Funding

Ministry of Science and Technology of Taiwan.

Disclosure

All authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.