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Poster display session: Basic science, Endocrine tumours, Gastrointestinal tumours - colorectal & non-colorectal, Head and neck cancer (excluding thyroid), Melanoma and other skin tumours, Neuroendocrine tumours, Thyroid cancer, Tumour biology & pathology

2787 - The Clinical Features and Genomic Landscape of Elevated Microsatellite Alterations at Selected Tetranucleotide Repeats (EMAST) in Patients with Colorectal Cancer

Date

21 Oct 2018

Session

Poster display session: Basic science, Endocrine tumours, Gastrointestinal tumours - colorectal & non-colorectal, Head and neck cancer (excluding thyroid), Melanoma and other skin tumours, Neuroendocrine tumours, Thyroid cancer, Tumour biology & pathology

Topics

Targeted Therapy

Tumour Site

Colon and Rectal Cancer

Presenters

Ming-Huang Chen

Citation

Annals of Oncology (2018) 29 (suppl_8): viii150-viii204. 10.1093/annonc/mdy281

Authors

M. Chen1, S. Chang2, P. Lin3, S. Yang4, C. Lin4, Y. Lan4, H. Lin4, C. Lin5, W. Liang6, W. Chen4, J. Jiang7, J. Lin7

Author affiliations

  • 1 Department Of Oncology, Taipei Veterans General Hospital, 11211 - Taipei/TW
  • 2 Department Of Surgery, Faculty Of Medicine,, National Yang-Ming University, 112 - Taipei/TW
  • 3 Department Of Health And Welfare, University of Taipei, 112 - Taipei/TW
  • 4 Surgery, Taipei Veterans General Hospital, 11217 - Taipei/TW
  • 5 Division Of Genomic Medicine, National Health Research Institutes, 11217 - Taipei/TW
  • 6 Pathology, Taipei Veterans General Hospital, 11217 - Taipei/TW
  • 7 Taipei Veterans General Hospital, Department of Surgery, 112 - Taipei/TW
More

Abstract 2787

Background

The form of microsatellite instability (MSI) affecting tetranucleotide repeats known as EMAST (elevated microsatellite alterations at selected tetranucleotide repeats) has emerged as a new potential biomarker in multiple cancers. In colorectal cancer (CRC), the clinical implications and mutation spectrum of EMAST mutations remain inconclusive.

Methods

We evaluated 1,505 CRC cases using five EMAST markers (D20S82, D20S85, D8S321, D9S242 and MYCL1) and the Bethesda panel of MSI markers. Most commonly mutations involved in CRCs were identified by MassArray Assay and DNA repair genes were analyzed by Next-Generation Sequencing (NGS). Clinical characteristics and prognostic relevance were correlated with EMAST. SPSS software (version 16.0) was used to perform all statistical analyses.

Results

Tumors with EMAST-positivity were detected in 159 (10.6%) out of 1,505 CRC cases and associated with unique clinical features including female predominance (p = 0.017), higher prevalence of proximal colon tumors (p < 0.001), early stage tumors (p = 0.002), poorly differentiated tumors (p < 0.001), mucinous histology (p = 0.001), and MSI (p < 0.001) and higher incidence of mutations in PI3KCA (p = 0.003), BRAF (p < 0.001), TGFßR (p < 0.001), PTEN (p = 0.001), and AKT1 (p = 0.04) compared with EMAST-negative tumors. Compared with EMAST-positive alone or MSI-H alone tumors, EMAST-positive MSI-H tumors had higher rates of MSH6, MSH3, PMS2, and EXO1 gene mutation (p < 0.001, p = 0.005, p = 0.001, and p = 0.027) and MLH1, MSH6, and EXO1 gene mutation (p = 0.019, p = 0.005, and p = 0.046), respectively. Finally, EMAST-positivity was a good prognostic indicator in early stage CRC (p = 0.002) but not in late CRC (p = 0.920).

Conclusions

The EMAST defines a unique molecular subtype of CRC.

Clinical trial identification

Legal entity responsible for the study

Taipei Veterans General Hospital.

Funding

Taipei Veterans General Hospital, Ministry of Science and Technology.

Editorial Acknowledgement

Disclosure

All authors have declared no conflicts of interest.

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