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Poster display session: Basic science, Endocrine tumours, Gastrointestinal tumours - colorectal & non-colorectal, Head and neck cancer (excluding thyroid), Melanoma and other skin tumours, Neuroendocrine tumours, Thyroid cancer, Tumour biology & pathology

1265 - Targeted Therapy based on the Genetic Alterations Prolongs the Progression-free Survival of Patients with Advanced Biliary Tract Cancer

Date

21 Oct 2018

Session

Poster display session: Basic science, Endocrine tumours, Gastrointestinal tumours - colorectal & non-colorectal, Head and neck cancer (excluding thyroid), Melanoma and other skin tumours, Neuroendocrine tumours, Thyroid cancer, Tumour biology & pathology

Topics

Targeted Therapy

Tumour Site

Hepatobiliary Cancers

Presenters

Feiling Feng

Citation

Annals of Oncology (2018) 29 (suppl_8): viii205-viii270. 10.1093/annonc/mdy282

Authors

F. Feng1, Q. Cheng1, D. Zhang2, B. Li1, H. Qin2, C. Xu1, M. Han2, Y. Yu1, Z. Li1, J. Li2, Z. Qiu1, L. Xiong2, C. Liu1, F. Li2, B. Yi1, X. Jiang1

Author affiliations

  • 1 Department Of Biliary I, Eastern Hepatobiliary Surgical Hospital(EHSH),2nd Military Medical University, 200438 - Shanghai/CN
  • 2 Research And Development Institute Of Precision Medicine, 3D Medicine Inc., Shanghai, China., 201114 - Shanghai/CN

Resources

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Abstract 1265

Background

Targeted therapy based on a certain genetic alteration has been proven to be an effective treatment in some kinds of cancers. We aimed to evaluate the clinical efficacy of personalized targeted therapy for advanced biliary tract cancer (BTC) patients based on individual actionable mutations.

Methods

In this single-center study, the targeted next generation sequencing (NGS) was employed for consecutive 49 patients with BTC and the recommendation of biologic agent was offered according to most potentially targetable genetic alteration of each person. Among 32 patients with stage IV and R2 resection, 21 patients underwent conventional chemotherapy (mGEMOX), while the rest 11 patients received a personalized targeted agent alternative to chemotherapy. The progression-free survival (PFS), overall survival (OS) and disease control rate (DCR) were assessed.

Results

The genomic landscape of 49 patients demonstrated that TP53 (n = 31, 63.3%) variation was most prevalent and was followed by KRAS (n = 12, 24.5%), ARID1A (n = 6, 12.2%), PIK3CA (n = 6, 12.2%), SMAD4 (n = 6, 12.2%), CDKN2A (n = 5, 10.2%) and ERBB4 (n = 5, 10.2%). Further analysis of copy number alterations (CNAs) showed low recurrent amplified genes, such as PIK3CA, SMAD4, FGFR3, SRC, PIK3R2, CDK4, ERBB2, and CDK6. After a median follow-up of 12 months, the targeted group had a significant prolonged PFS (4.5 months vs. 1.5 months, P = 0.014) and a trend of prolonged OS (12.9 months vs. 4.1 months, P = 0.104) in comparison with the chemotherapy group. The DCR in targeted group was a little higher but without significance (63.6% vs. 33.3%, P = 0.142). In addition, the ratio of patients with more than Grade 2 treatment-related toxicities in the targeted therapy group was a little higher compared to that of chemotherapy group but without significance (36.4% vs. 19.0%, P = 0.397).

Conclusions

This mono-center small cohort study suggested that personalized targeted therapy for advanced BTC patients based on individual most potentially actionable mutation detected by NGS, which offered such patients a longer PFS and trends of better OS and DCR, could be an option alternative to conventional chemotherapy.

Clinical trial identification

Legal entity responsible for the study

Shanghai Eastern Hepatobiliary Surgery Hospital, Second Military Medical University, Shanghai, China.

Funding

Science and Technology Commission of Shanghai Municipality (NO. 15411951900), National Natural Science Foundation of China (NO. 81472280), Special Fund on the Application and Transformation of Precision Medicine in Second Military Medical University (NO.2017JZ11).

Editorial Acknowledgement

There is not any editorial assistance for this abstract provided by a third party.

Disclosure

L. Xiong: Chairman, Chief Executive Officer: 3D Medicine, Inc. F. Li: Vice President: 3D Medicine, Inc. J.Y. Li, H. Qin, M. Han, D. Zhang: Employee: 3D Medicine, Inc. All other authors have declared no conflicts of interest.

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