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Poster display session: Breast cancer - early stage, locally advanced & metastatic, CNS tumours, Developmental therapeutics, Genitourinary tumours - prostate & non-prostate, Palliative care, Psycho-oncology, Public health policy, Sarcoma, Supportive care

1484 - Stereotactic Radiosurgery (SRS) for brain metastases (BM) from breast cancer (BC) – a single centre experience of factors influencing survival

Date

22 Oct 2018

Session

Poster display session: Breast cancer - early stage, locally advanced & metastatic, CNS tumours, Developmental therapeutics, Genitourinary tumours - prostate & non-prostate, Palliative care, Psycho-oncology, Public health policy, Sarcoma, Supportive care

Topics

Surgical Oncology;  Radiation Oncology

Tumour Site

Breast Cancer

Presenters

Tom Wilson

Citation

Annals of Oncology (2018) 29 (suppl_8): viii90-viii121. 10.1093/annonc/mdy272

Authors

T. Wilson1, T. Robinson1, C. Macfarlane2, T. Spencer3, C. Herbert3, J. Braybrooke1

Author affiliations

  • 1 Medical Oncology, University Hospitals Bristol NHS Trust Bristol Haematology and Oncology Centre, BS2 8ED - Bristol/GB
  • 2 Oncology, University Hospital Southampton NHS Foundation Trust, SO16 6YD - Southampton/GB
  • 3 Clinical Oncology, University Hospitals Bristol NHS Trust Bristol Haematology and Oncology Centre, BS2 8ED - Bristol/GB

Resources

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Abstract 1484

Background

BM occur in up to 30% of patients with BC. SRS can be considered as an alternative to surgery or whole brain radiotherapy for control of single or multiple metastases. Factors that could predict overall survival (OS) and guide treatment choice have not been clearly defined.

Methods

Data for consecutive patients with BC treated with SRS between November 2013 and August 2017 in a single tertiary referral centre were collected to assess factors that might predict survival. T-tests, Kaplan Meier and log-rank methods were used for statistical analysis.

Results

93 patients (pts) received SRS, of whom 32 were alive when data was censored on 1/2/18. The median overall survival (OS) post SRS was 14.0 mths (95% C.I +/- 2.4mths) with a significant correlation between age and OS (53 pts ≤60yr, OS = 18.1mths; 43pts >60y OS = 10.3mths, r=-0.21, p = 0.04). There were 31 ER+/HER2- pts (OS 15.2mths), 14 ER+/HER2+ pts (OS 31.9mths), 30 ER-/HER2+ pts (OS 22.8mths) and 16 Triple negative (TN) pts (OS 8.5mths). OS was significantly better for pts with HER2+ disease (p = 0.0018) with the poorest survival in pts with TN cancer (p = <0.0001). 39pts had 1 lesion treated (OS 16.2mths), 35pts had 2-5 lesions treated (OS 9.7mths), 19 pts had ≥6 lesions (OS 19.2mths). Whilst the number of lesions treated did not correlate with OS (r=-0.095, p = 0.37) a larger volume of tumour treated (>10cm3) was associated with worse survival (r=-0.253, p = 0.01). At the time of SRS 17pts had no other systemic disease (OS 13.7mths). When present, control of systemic disease outside the brain was associated with improved OS (36 pts stable systemic disease OS = 20mths, 32 pts progressive systemic disease (PSD) OS = 9.7mths; p = 0.0013). 49 pts had known disease status at death, 17 had PSD, 16 had progressive CNS disease (PCD), 13 had PSD + PCD and 3 had no progression.

Conclusions

Age, receptor status, control of systemic disease, total tumour volume treated, but not the number of lesions appears to influence survival following SRS treatment of BM in BC. Our results are consistent with other series with worse outcomes seen in older patients and those with TN cancers. In addition to treating BM, control of systemic disease outside the brain remains important in prolonging survival.

Clinical trial identification

Legal entity responsible for the study

Jeremy Braybrooke.

Funding

Has not received any funding.

Editorial Acknowledgement

Disclosure

All authors have declared no conflicts of interest.

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