Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster display session: Basic science, Endocrine tumours, Gastrointestinal tumours - colorectal & non-colorectal, Head and neck cancer (excluding thyroid), Melanoma and other skin tumours, Neuroendocrine tumours, Thyroid cancer, Tumour biology & pathology

5044 - Non-invasive genotyping and monitoring of tumor evolution in locally advanced rectal cancer (LARC) patients using circulating tumor DNA (ctDNA)

Date

21 Oct 2018

Session

Poster display session: Basic science, Endocrine tumours, Gastrointestinal tumours - colorectal & non-colorectal, Head and neck cancer (excluding thyroid), Melanoma and other skin tumours, Neuroendocrine tumours, Thyroid cancer, Tumour biology & pathology

Topics

Translational Research

Tumour Site

Colon and Rectal Cancer

Presenters

Sinead Toomey

Citation

Annals of Oncology (2018) 29 (suppl_8): viii150-viii204. 10.1093/annonc/mdy281

Authors

S. Toomey1, A. Sartori2, D. Irwin3, S. Hummel4, A. Carr5, C.L. Lee6, P. Armstrong5, A. Farrelly7, S. El-Masry8, D. McNamara9, P.G. Morris10, L. Grogan10, O.S. Breathnach10, L. O'Sullivan11, S. Bradshaw12, A.N.A.M. Rashed13, R. Smyth5, J. Workman5, B. O'Neill14, B. Hennessy10

Author affiliations

  • 1 Molecular Medicine, Royal College of Surgeons in Ireland, 2 - Dublin/IE
  • 2 Scientific Affairs, Agena Biosciences, 22761 - Hamburg/DE
  • 3 Agena Bioscience, Agena Biosciences, 4006 - Brisbane/AU
  • 4 Agena Bioscience, Agena Biosciences, 92121 - San Diego/US
  • 5 Molecular Medicine, Royal College of Surgeons in Ireland, D09YD60 - Dublin/IE
  • 6 Medical Oncology, St Vincents University Hospital, 4 - Dublin/IE
  • 7 Oncology Molecular Medicine, Royal College of Surgeons in Ireland, Beaumont Hospital, 9 - Dublin/IE
  • 8 Surgery, Our Lady of Lourdes Hospital, Drogheda, 0000 - Drogheda/IE
  • 9 Surgery, Beaumont Hospital, D09YD60 - Dublin/IE
  • 10 Medical Oncology, Beaumont Hospital, 9 - Dublin/IE
  • 11 Cancer Trials Ireland, Cancer Trials Ireland, 0000 - Dublin/IE
  • 12 Radiation oncology, St Luke's Radiation oncology Network, 0000 - Dublin/IE
  • 13 Oncology, Our Lady of Lourdes Hospital, Drogheda/IE
  • 14 Radiation oncology, St. Luke's Radiation oncology Network, D09YD60 - Dublin/IE

Resources

Login to access the resources on OncologyPRO.

If you do not have an ESMO account, please create one for free.

Abstract 5044

Background

There is limited information on the feasibility and clinical potential of ctDNA analysis in non-metastatic rectal cancer. We assessed whether detection and analysis of plasma ctDNA could help monitor tumor evolution during neoadjuvant chemoradiotherapy (NACRT) treatment for LARC.

Methods

27 LARC patients who were enrolled on the CTRIAL-IE (ICORG) 12-38 TRI-LARC clinical trial (NCT02151019) and received NACRT prior to surgery, had samples for ctDNA analysis taken at baseline pre-NACRT, during week 3 of radiotherapy (RT), during the final week of RT, prior to surgery, and 3-12 months post-surgery. DNA from baseline biopsy samples was genotyped for 86 hotspot mutations in BRAF, EGFR, KRAS, NRAS and PIK3CA using the iPLEX™ HS Colon Panel on the MassARRAY® System (Agena Bioscience). The UltraSEEK™ Colon Panel (Agena Bioscience) was used to track 107 hotspot mutations in serial ctDNA samples.

Results

At least one mutation was identified in 67% (18/27) of baseline biopsy samples. 15 patients (56%) had a KRAS mutation. Identical mutations were found in baseline ctDNA of 11/15 patients (73%). The median KRAS mutant allele frequency (MAF) in baseline ctDNA samples was 0.9% (range 0.1-2%). This significantly decreased over treatment (0.15% week 3 RT, 0.35% final week RT, 0.1% prior to surgery) (p < 0.05). 10 patients had a KRAS mutation in their baseline ctDNA that was not detected in their biopsy, with a median MAF of 0.3% (range 0.1-1.3%). Mutations in NRAS and PIK3CA were identified in 3/27 (11%) and 2/27 (7%) of patients, respectively. NRAS and PIK3CA mutations were identified in the ctDNA of 2/3 (67%) and 1/2 (50%) patients. Post-operative ctDNA samples were available for 13 patients and residual mutations were detected in 10 patients (77%), 3 of whom (30%) had recurrence at median follow up of 20.1 months. There was no recurrence in any patient with negative ctDNA post-surgery.

Conclusions

ctDNA can identify clinically relevant biomarkers and could be used as a minimally invasive alternative to repeated tumor biopsies to monitor tumor evolution. ctDNA detection after resection may provide evidence of residual disease and could identify patients at high risk of recurrence.

Clinical trial identification

NCT02151019.

Legal entity responsible for the study

Cancer Trials Ireland.

Funding

St. Luke's Institute of Cancer Research.

Editorial Acknowledgement

Disclosure

A. Sartori, D. Irwin: Employee, stock owner: Agena Biosciences. S. Hummel: Employee: Agena Biosciences. All other authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.