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Poster display session: Biomarkers, Gynaecological cancers, Haematological malignancies, Immunotherapy of cancer, New diagnostic tools, NSCLC - early stage, locally advanced & metastatic, SCLC, Thoracic malignancies, Translational research

5378 - Hybrid capture NGS reliably detects a spectrum of clinically significant genetic aberrations in both, primary diagnostics and the relapse scenario.

Date

20 Oct 2018

Session

Poster display session: Biomarkers, Gynaecological cancers, Haematological malignancies, Immunotherapy of cancer, New diagnostic tools, NSCLC - early stage, locally advanced & metastatic, SCLC, Thoracic malignancies, Translational research

Presenters

Stefanie Schatz

Citation

Annals of Oncology (2018) 29 (suppl_8): viii493-viii547. 10.1093/annonc/mdy292

Authors

S. Schatz1, M. Falk1, R. Menon2, L.C. Heukamp2, J. Roeper3, F. Griesinger4, M. Tiemann5

Author affiliations

  • 1 Hematopathology Hamburg, Hematopathology Hamburg, 22547 - Hamburg/DE
  • 2 Neo New Oncology, NEO New Oncology, Cologne/DE
  • 3 Pius Hospital Oldenburg, University Of Oldenburg, Pius Hospital Oldenburg, University of Oldenburg, Oldenburg/DE
  • 4 Pius Hospital Oldenburg, Pius Hospital Oldenburg, 26121 - Oldenburg/DE
  • 5 Hematopathology Hamburg, Hematopathology Hamburg, Hamburg/DE

Resources

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Abstract 5378

Background

The purpose of this study was to analyze the frequency and type of genetic alterations in tyrosine kinase inhibitor (TKI)-naïve vs. TKI-treated NSCLC patients. We report a spectrum of resistance mechanisms upon TKI treatment in liquid biopsies using a comprehensive hybrid capture NGS test (HC-NGS).

Methods

We retrospectively collected mutational data of 200 solid NSCLC biopsies of treatment-naïve patients that were routinely tested for clinically relevant mutations by HC-NGS at our institution. In addition, we analyzed mutational data from 367 liquid biopsies of NSCLC patients whose disease progressed under treatment. Solid tumor biopsies and liquid biopsies were tested using a 39-gene HC-NGS panel including 39 clinically relevant genes (NEOselect, LOD 3.0%; NEOliquid, LOD 0.1%; NEO New Oncology GmbH, Cologne).

Results

Primary HC-NGS diagnostics in TKI -aïve patients revealed 12% (24/200) TKI-sensitive EGFR mutations, 5.0% (10/200) Alk/ROS/RET translocations, 0.5% (1/200) BRAF V600E, and in one patient a MET Exon 14 skipping mutation (0.5%, 1/200). Further, a variety of clinically relevant mutations were detected, among others, prognostically relevant TP53 mutations at a frequency of 52.0% (104/200). In addition, we tested 367 liquid biopsies from relapse patients. 30.9% (113/367) pts had an activating EGFR mutation and therefore presumably received EGFR TKI treatment. Of the 113 pts, 39.8% (45/113) developed a T790M resistance mutation and 1.8% (2/113) an additional MET amplification.

Conclusions

HC-NGS allows for comprehensive analysis of somatic tumor aberrations in the primary diagnostic setting as well as in the relapse scenario. Mechanisms of primary oncogenic activation as well as mechanisms of resistance are heterogeneous and include point mutations, translocations and gene amplifications. Therefore, HC-NGS should be used for diagnosis in both, the primary and the relapse setting.

Clinical trial identification

Legal entity responsible for the study

Hematopathology Hamburg.

Funding

Has not received any funding.

Editorial Acknowledgement

Disclosure

L.C. Heukamp: Affiliations: Hematopathology Hamburg, NEO New Oncology. M. Tiemann: Anstellungsverhaltnis oder Führungsposition: CEO Institut für Heamatopathologie Hamburg Beratungs- bzw. Gutachtertätigkeit; Advisory boards: Novartis, Boehringer, Roche, AstraZeneca; Honorare erhalten von: Vortragshonorare: Novartis, Boehringer, Roche, AstraZeneca. All other authors have declared no conflicts of interest.

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